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Emma Weisblatt

Researcher at University of Cambridge

Publications -  16
Citations -  1986

Emma Weisblatt is an academic researcher from University of Cambridge. The author has contributed to research in topics: Autism & Autism spectrum disorder. The author has an hindex of 8, co-authored 15 publications receiving 1883 citations. Previous affiliations of Emma Weisblatt include University College London.

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Mapping autism risk loci using genetic linkage and chromosomal rearrangements

Peter Szatmari, +139 more
- 01 Mar 2007 - 
TL;DR: Linkage and copy number variation analyses implicate chromosome 11p12–p13 and neurexins, respectively, among other candidate loci, highlighting glutamate-related genes as promising candidates for contributing to ASDs.
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Speech-in-Noise Perception in High-Functioning Individuals with Autism or Asperger's Syndrome.

TL;DR: The results suggest that the speech-in-noise perception difficulties experienced by individuals with autism may be due, in part, to a reduced ability to integrate information from glimpses present in the temporal dips in the noise.
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Towards an understanding of the mechanisms of weak central coherence effects: experiments in visual configural learning and auditory perception.

TL;DR: It is proposed that perception operates to enhance the representation of individual perceptual features but that this does not impact adversely on representations that involve integration of features.
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Chromosome 15q11-13 abnormalities and other medical conditions in individuals with autism spectrum disorders.

TL;DR: The overall rate of possibly causal medical and cytogenetic conditions in children with autism spectrum disorders was low and no different from the rate of disorder inChildren with other developmental/neuropsychiatric disorders that attended the same clinics.
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Auditory temporal-envelope processing in high-functioning children with Autism Spectrum Disorder.

TL;DR: Low-pass filter model estimates of temporal-envelope resolution and temporal-processing efficiency showed significant differences between the groups for modulation-depth threshold values at low modulation rates, consistent with ASD individuals having reduced processing efficiency of temporal modulations.