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Fujiko Irie

Researcher at Dokkyo Medical University

Publications -  67
Citations -  4051

Fujiko Irie is an academic researcher from Dokkyo Medical University. The author has contributed to research in topics: Hazard ratio & Population. The author has an hindex of 26, co-authored 63 publications receiving 3451 citations. Previous affiliations of Fujiko Irie include University of Tsukuba.

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Association between Body-Mass Index and Risk of Death in More Than 1 Million Asians

TL;DR: Underweight was associated with a substantially increased risk of death in all Asian populations, however, the excess risk of died was seen among East Asians but not among Indians and Bangladeshis.
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Decline in Estimated Glomerular Filtration Rate and Subsequent Risk of End-Stage Renal Disease and Mortality

TL;DR: Declines in estimated GFR smaller than a doubling of serum creatinine concentration occurred more commonly and were strongly and consistently associated with the risk of ESRD and mortality, supporting consideration of lesser declines in Estimated GFR (such as a 30% reduction over 2 years) as an alternative end point for CKD progression.
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Body Mass Index and Diabetes in Asia: A Cross-Sectional Pooled Analysis of 900,000 Individuals in the Asia Cohort Consortium

TL;DR: The positive association between BMI and diabetes prevalence was present in all cohorts and in all subgroups of the study population, although the association was stronger in individuals below age 50 at baseline.
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The relationships of proteinuria, serum creatinine, glomerular filtration rate with cardiovascular disease mortality in Japanese general population

TL;DR: For individuals with proteinuria combined by hypercreatininemia or reduced GFR, the risk of CVD death was two-foldHigher in men and 4-6-fold higher in women compared to those without proteinuria and with normal creatinine level or GFR.
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Glomerulonephritis associated with MRSA infection: A possible role of bacterial superantigen

TL;DR: It is speculated that post-MRSA glomerulonephritis may be induced by superantigens causing production of high levels of cytokines, and polyclonal activation of IgG and IgA, as well as the formation of ICs containing IgA and IgG in the circulation result in development of glomeral onephritis and vasculitis.