G
Giorgio Battaglia
Researcher at University of Strathclyde
Publications - 75
Citations - 3677
Giorgio Battaglia is an academic researcher from University of Strathclyde. The author has contributed to research in topics: Cortical dysplasia & Epilepsy. The author has an hindex of 26, co-authored 74 publications receiving 3258 citations.
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Journal ArticleDOI
The clinicopathologic spectrum of focal cortical dysplasias: A consensus classification proposed by an ad hoc Task Force of the ILAE Diagnostic Methods Commission
Ingmar Blümcke,Maria Thom,Eleonora Aronica,Dawna D. Armstrong,Harry V. Vinters,André Palmini,Thomas S. Jacques,Giuliano Avanzini,A. James Barkovich,Giorgio Battaglia,Albert J. Becker,Carlos Cepeda,Fernando Cendes,Nadia Colombo,Peter B. Crino,J. Helen Cross,Olivier Delalande,François Dubeau,John S. Duncan,Renzo Guerrini,Philippe Kahane,Gary W. Mathern,Imad Najm,Cigdem Ozkara,Charles Raybaud,Alfonso Represa,Steven N. Roper,Noriko Salamon,Andreas Schulze-Bonhage,Laura Tassi,Annamaria Vezzani,Roberto Spreafico +31 more
TL;DR: Focal cortical dysplasias (FCD) are localized regions of malformed cerebral cortex and are very frequently associated with epilepsy in both children and adults.
Journal ArticleDOI
Mutations in the mitochondrial protease gene AFG3L2 cause dominant hereditary ataxia SCA28
Daniela Di Bella,Federico Lazzaro,Alfredo Brusco,Massimo Plumari,Giorgio Battaglia,Annalisa Pastore,Adele Finardi,Claudia Cagnoli,Filippo Tempia,Marina Frontali,Liana Veneziano,Tiziana Sacco,Enrica Boda,Alessandro Brussino,Florian Bonn,Barbara Castellotti,Silvia Baratta,Caterina Mariotti,Cinzia Gellera,Valentina Fracasso,Stefania Magri,Thomas Langer,Paolo Plevani,Stefano Di Donato,Marco Muzi-Falconi,Franco Taroni +25 more
TL;DR: This work identifies AFG3L2 as a novel cause of dominant neurodegenerative disease and indicates a previously unknown role for this component of the mitochondrial protein quality control machinery in protecting the human cerebellum against neurodegenersation.
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Periventricular nodular heterotopia: Classification, epileptic history, and genesis of epileptic discharges
Giorgio Battaglia,Luisa Chiapparini,Silvana Franceschetti,Elena Freri,Laura Tassi,Stefania Bassanini,Flavio Villani,Roberto Spreafico,Ludovico D'Incerti,Tiziana Granata +9 more
TL;DR: Clinical, MRI, and electrophysiologic findings in 54 PNH patients are analyzed to reevaluate the classification of PNH, relate the anatomic features to epileptic outcome, and ascertain the contribution of P NH nodules to the onset of epileptic discharges.
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Flexible endoscopic treatment for Zenker's diverticulum: a systematic review and meta-analysis.
Sauid Ishaq,Sauid Ishaq,Cesare Hassan,Alessandro Antonello,Keeley Tanner,Cristina Bellisario,Giorgio Battaglia,Andrea Anderloni,Loredana Correale,Prateek Sharma,Todd H. Baron,Alessandro Repici,Alessandro Repici +12 more
TL;DR: FESD is a feasible, safe, and effective treatment for symptomatic ZD, with low adverse event and recurrence rates, including an in-depth evaluation of its efficacy, safety, and limitations.
Journal ArticleDOI
Misplaced NMDA receptors in epileptogenesis contribute to excitotoxicity.
Angelisa Frasca,Marlien W. Aalbers,Federica Frigerio,Fabio Fiordaliso,Monica Salio,Marco Gobbi,Alfredo Cagnotto,Fabrizio Gardoni,Giorgio Battaglia,Govert Hoogland,Monica Di Luca,Annamaria Vezzani +11 more
TL;DR: Pharmacological blockade of NR2B-containing NMDARs by ifenprodil administration during epileptogenesis significantly reduced pyramidal cell loss in the hippocampus, showing that the observed post-translational and cellular changes ofNR2B subunit contribute to excitotoxicity.