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Joachim Heinrich

Researcher at Ludwig Maximilian University of Munich

Publications -  1327
Citations -  88485

Joachim Heinrich is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topics: Population & Asthma. The author has an hindex of 136, co-authored 1309 publications receiving 76887 citations. Previous affiliations of Joachim Heinrich include Politehnica University of Bucharest & Universidade Nova de Lisboa.

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Glutamine/histidine polymorphism in apo A-IV affects plasma concentrations of lipoprotein(a) and fibrin split products in coronary heart disease patients.

TL;DR: A hitherto unappreciated role of the apo A-IV gene or a closely linked locus for the regulation of Lp(a) metabolism and hemostasis and also possibly for atherosclerosis and thrombosis is indicated.
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Gas cooking is associated with small reductions in lung function in children

TL;DR: Under current housing conditions, gas cooking is associated with only small reductions in lung function, and there was some indication that allergic children were more affected by gas cooking.
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Health-related quality of life and chronic obstructive pulmonary disease in early stages – longitudinal results from the population-based KORA cohort in a working age population

TL;DR: It is indicated that it is important to prevent subjects with airflow limitation from progression to higher grades in COPD patients over ten years and awareness of HRQL impairments in early stages is important for offering early interventions in order to maintain high HRQL.
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Prospective relevance of dietary patterns at the beginning and during the course of primary school to the development of body composition

TL;DR: In this paper, the authors identify dietary patterns at the beginning of and during the primary school period and examine their relevance to the development of body composition, using reduced rank regression (RRR) to directly extract patterns explaining variation in changes in BMI and FMI between ages 6 and 7 years and ages 10 and 11 years.
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Source category-specific PM2.5 and urinary levels of Clara cell protein CC16. The ULTRA study.

TL;DR: PM2.5 from combustion sources increases epithelial barrier permeability in lungs and is associated with urinary CC16 in Helsinki, Finland, but not in the other two ULTRA cities.