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Joachim Heinrich

Researcher at Ludwig Maximilian University of Munich

Publications -  1327
Citations -  88485

Joachim Heinrich is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topics: Population & Asthma. The author has an hindex of 136, co-authored 1309 publications receiving 76887 citations. Previous affiliations of Joachim Heinrich include Politehnica University of Bucharest & Universidade Nova de Lisboa.

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Exposure to endotoxin decreases the risk of atopic eczema in infancy: A cohort study

TL;DR: The hygiene hypothesis that exposure to high concentrations of endotoxin very early in life might protect against the development of atopic eczema within the first 6 months of life, along with an increased prevalence of nonspecific respiratory diseases is supported.

Genome-wide associations for birth weight and correlations with adult disease

Momoko Horikoshi, +162 more
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Genome Analyses of >200,000 Individuals Identify 58 Loci for Chronic Inflammation and Highlight Pathways that Link Inflammation and Complex Disorders.

Symen Ligthart, +286 more
TL;DR: In this article, the authors performed two genome-wide association studies (GWASs), on HapMap and 1000 Genomes imputed data, of circulating amounts of CRP by using data from 88 studies comprising 204,402 European individuals.
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SNPs of the FADS gene cluster are associated with polyunsaturated fatty acids in a cohort of patients with cardiovascular disease.

TL;DR: The observed strong association of FADS gene polymorphisms with the levels of arachidonic acid, which is a precursor of molecules involved in inflammation and immunity processes, suggests that SNPs of the FADS1 and FADS2 gene region are worth studying in diseases related to inflammatory conditions or alterations in the concentration of PUFAs.
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Long-Term Exposure to Ambient Air Pollution and Cardiopulmonary Mortality in Women

TL;DR: Living close to major roads and chronic exposure to NO2 and PM10 may be associated with an increased mortality due to cardiopulmonary causes.