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A. von Eckardstein

Researcher at University of Zurich

Publications -  84
Citations -  4290

A. von Eckardstein is an academic researcher from University of Zurich. The author has contributed to research in topics: Cholesterol & Apolipoprotein B. The author has an hindex of 38, co-authored 84 publications receiving 4112 citations. Previous affiliations of A. von Eckardstein include University of Münster & Leibniz Association.

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Journal Article

The emergence of triglycerides as a significant independent risk factor in coronary artery disease

TL;DR: The Prospective Cardiovascular Münster (PROCAM) study showed that fasting levels of triglycerides were an independent risk factor for CHD events, irrespective of serum levels of high density lipoprotein cholesterol (HDL-C) or low density cholesterol (LDL-L), and that individuals with potentially atherogenic lipid profiles should be managed initially through the introduction of lifestyle changes.
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Meta-analysis identifies multiple loci associated with kidney function–related traits in east Asian populations

Yukinori Okada, +414 more
- 01 Aug 2012 - 
TL;DR: A meta-analysis of genome-wide association studies for kidney function–related traits, including 71,149 east Asian individuals from 18 studies in 11 population-, hospital- or family-based cohorts, conducted as part of the Asian Genetic Epidemiology Network (AGEN), identified 17 loci newly associated with kidney function-related traits.
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A plasma lipoprotein containing only apolipoprotein E and with gamma mobility on electrophoresis releases cholesterol from cells.

TL;DR: The results strongly indicate the presence in human plasma of a lipoprotein containing only apoE, gamma-LpE, which is secreted by peripheral cells and is a potent acceptor of cell-derived cholesterol.
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The CAG repeat polymorphism in the androgen receptor gene modulates body fat mass and serum concentrations of leptin and insulin in men

TL;DR: The pivotal role of this polymorphism in modulating androgen effects on cardiovascular risk factors is of a complex nature and implies that its clinical impact, similar to that of androgens, is dependent on exogenous cofactors.
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Effects of age, lipoproteins, and hemostatic parameters on the role of homocyst(e)inemia as a cardiovascular risk factor in men.

TL;DR: It is concluded that hyperhomocyst(e)inemia is an independent coronary risk factor and that its interrelation with fibrinogen levels merits further study.