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Ke Xu

Researcher at Yale University

Publications -  129
Citations -  9268

Ke Xu is an academic researcher from Yale University. The author has contributed to research in topics: Population & Medicine. The author has an hindex of 37, co-authored 103 publications receiving 8354 citations. Previous affiliations of Ke Xu include Veterans Health Administration & National Institutes of Health.

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Interaction between a functional MAOA locus and childhood sexual abuse predicts alcoholism and antisocial personality disorder in adult women.

TL;DR: MAOA seems to moderate the impact of childhood trauma on adult psychopathology in female subjects in the same way as previously shown among male subjects.
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Association between ADORA2A and DRD2 polymorphisms and caffeine-induced anxiety.

TL;DR: Ass associations between self-reported anxiogenic effects of caffeine and variation in the genes for A2A (ADORA2A) and DRD2 (DRD2) receptors are examined to provide support for an association between an ADORA 2A polymorphism and self- reported anxiety after a moderate dose of caffeine.
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Association of the G1947A COMT (Val108/158Met) gene polymorphism with prefrontal P300 during information processing

TL;DR: The association of the frontal P300 amplitude with the G1947A COMT genotype further emphasizes the functional role of this SNP and suggests that the amount of noise in prefrontal neural networks during information processing might be in part under genetic control, which is mediated by dopamine.
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Nuclear factor κB is a critical determinant in N-methyl-D-aspartate receptor-mediated neuroprotection

TL;DR: Nuclear extracts prepared from neurons treated with NMDA and the double‐stranded NF‐κB oligonucleotide showed reduced DNA binding activity to the target sequence, supporting the idea that NF‐kkB may be involved in the transcriptional activation of the BDNF gene.
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DRD2 promoter region variation as a predictor of sustained response to antipsychotic medication in first-episode schizophrenia patients

TL;DR: Findings suggest that variation in the D(2) receptor gene can, in part, explain variations in the timing of clinical response to antipsychotics in patients with first-episode schizophrenia.