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Kim B. Ritchie

Researcher at University of South Carolina Beaufort

Publications -  51
Citations -  5814

Kim B. Ritchie is an academic researcher from University of South Carolina Beaufort. The author has contributed to research in topics: Acropora & Coral reef. The author has an hindex of 28, co-authored 47 publications receiving 5347 citations. Previous affiliations of Kim B. Ritchie include University of Florida & University of North Carolina at Chapel Hill.

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Journal ArticleDOI

Regulation of microbial populations by coral surface mucus and mucus-associated bacteria

TL;DR: Coral mucus inhibited antibiotic activity and pigment production in a poten- tially invasive bacterium, illustrating that coral mucus may inactivate mechanisms used for bacterial niche establishment and suggesting a microbial contribution to the antibacterial activity described for coral mucUS.
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Caribbean corals in crisis: record thermal stress, bleaching, and mortality in 2005.

C. Mark Eakin, +70 more
- 15 Nov 2010 - 
TL;DR: Comparison of satellite data against field surveys demonstrated a significant predictive relationship between accumulated heat stress (measured using NOAA Coral Reef Watch's Degree Heating Weeks) and bleaching intensity.
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The etiology of white pox, a lethal disease of the Caribbean elkhorn coral, Acropora palmata.

TL;DR: This report identifies a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox, the first time, to the authors' knowledge, that a bacterial species associated with the human gut has been shown to be a marine invertebrate pathogen.
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High frequency of horizontal gene transfer in the oceans.

TL;DR: It is demonstrated that the widespread capability of marine bacteria to participate in horizontal gene transfer (HGT) in coastal and oceanic environments may be the result of gene transfer agents (GTAs), viral-like particles produced by α-Proteobacteria.
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Interactions of TLC1 (Which Encodes the RNA Subunit of Telomerase), TEL1, and MEC1 in Regulating Telomere Length in the Yeast Saccharomyces cerevisiae

TL;DR: It is found that strains with both tel1 andtlc1 mutations have a delayed loss of cell viability compared to strains with the single tlc1 mutation, arguing that the role of Tel1p in telomere maintenance is not solely a direct activation of telomerase.