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Kohjiro Ueki

Researcher at University of Tokyo

Publications -  213
Citations -  27673

Kohjiro Ueki is an academic researcher from University of Tokyo. The author has contributed to research in topics: Diabetes mellitus & Insulin resistance. The author has an hindex of 60, co-authored 188 publications receiving 25194 citations. Previous affiliations of Kohjiro Ueki include Brigham and Women's Hospital.

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Adiponectin and adiponectin receptors in insulin resistance, diabetes, and the metabolic syndrome

TL;DR: The pathophysiology of adiponectin and adiponECTin receptors in insulin resistance, diabetes, and the metabolic syndrome is described and potential versatile therapeutic targets to combat obesity-linked diseases characterized by insulin resistance are described.
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CD8 + effector T cells contribute to macrophage recruitment and adipose tissue inflammation in obesity

TL;DR: The findings suggest that obese adipose tissue activates CD8+ T cells, which, in turn, promote the recruitment and activation of macrophages in this tissue, which supports the notion that CD8- T cells have an essential role in the initiation and propagation of adipose inflammation.
Journal Article

Report of the committee on the classification and diagnostic criteria of diabetes mellitus (revision for international harmonization of HbAle in Japan)

TL;DR: Diabetes mellitus is a group of diseases associated with various metabolic disorders, the main feature of which is chronic hyperglycemia due to insufficient insulin action, which can cause susceptibility to specific complications and also foster arteriosclerosis.
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Targeted disruption of AdipoR1 and AdipoR2 causes abrogation of adiponectin binding and metabolic actions.

TL;DR: Adenovirus-mediated expression of AdipoR1 and R2 in the liver of Lepr−/− mice increased AMP-activated protein kinase (AMPK) activation and peroxisome proliferator-activated receptor (PPAR)-α signaling pathways, respectively, and abolished adiponectin binding and actions, leading to insulin resistance and marked glucose intolerance in vivo.