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L. de Haan

Researcher at University of Amsterdam

Publications -  79
Citations -  3445

L. de Haan is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Schizophrenia & Psychosis. The author has an hindex of 29, co-authored 73 publications receiving 3032 citations. Previous affiliations of L. de Haan include University of Western Australia & St. Michael's Hospital.

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Subcortical brain volume abnormalities in 2028 individuals with schizophrenia and 2540 healthy controls via the ENIGMA consortium

T.G.M. van Erp, +66 more
- 01 Apr 2016 - 
TL;DR: Worldwide cooperative analyses of brain imaging data support a profile of subcortical abnormalities in schizophrenia, which is consistent with that based on traditional meta-analytic approaches, and validates that collaborative data analyses can readily be used across brain phenotypes and disorders.
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Childhood bullying and the association with psychosis in non-clinical and clinical samples: a review and meta-analysis.

TL;DR: This paper performed a meta-analysis on seven population-based studies, yielding unadjusted and adjusted odds ratios (ORs) of 2.7 [95% confidence interval (CI) 2.1-3.6] and 2.3 (95% CI 1.5 -3.4) respectively.
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Interventions to improve adherence to antipsychotic medication in patients with schizophrenia--a review of the past decade.

TL;DR: The literature between 2000 and 2009 was searched for randomized controlled trials which compared a psychosocial intervention with another intervention or with treatment as usual in patients with schizophrenia, with a large heterogeneity in design, adherence measures and outcome variables.
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White-matter markers for psychosis in a prospective ultra-high-risk cohort.

TL;DR: U HR subjects who later develop psychosis have differences in WM integrity, compared with UHR subjects who do not develop psychosis and to healthy controls, in brain areas associated with schizophrenia.
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Subjective experience and striatal dopamine D(2) receptor occupancy in patients with schizophrenia stabilized by olanzapine or risperidone.

TL;DR: Preliminary evidence that negative subjective experience is related to high D(2) receptor occupancy is provided, which may have implications for dosing strategies.