L
Lisa M. Muglia
Researcher at Cincinnati Children's Hospital Medical Center
Publications - 21
Citations - 1717
Lisa M. Muglia is an academic researcher from Cincinnati Children's Hospital Medical Center. The author has contributed to research in topics: Adenylyl cyclase & Glucocorticoid. The author has an hindex of 16, co-authored 20 publications receiving 1485 citations. Previous affiliations of Lisa M. Muglia include Vanderbilt University & Washington University in St. Louis.
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Journal ArticleDOI
Genetic Associations with Gestational Duration and Spontaneous Preterm Birth
Ge Zhang,Bjarke Feenstra,Jonas Bacelis,Xueping Liu,Lisa M. Muglia,Julius Juodakis,Daniel Miller,Nadia K. Litterman,Pan Pan Jiang,Laura Russell,David A. Hinds,Youna Hu,Matthew T. Weirauch,Xiaoting Chen,Arun R. Chavan,Günter P. Wagner,Günter P. Wagner,Mihaela Pavlicev,Mauris C. Nnamani,Jamie Maziarz,Minna K. Karjalainen,Minna K. Karjalainen,Mika Rämet,Mika Rämet,Verena Sengpiel,Frank Geller,Heather A. Boyd,Aarno Palotie,Aarno Palotie,Aarno Palotie,Allison Momany,Bruce Bedell,Kelli K Ryckman,Johanna M. Huusko,Johanna M. Huusko,Johanna M. Huusko,Carmy Forney,Leah C. Kottyan,Mikko Hallman,Mikko Hallman,Mikko Hallman,Kari Teramo,Ellen A. Nohr,George Davey Smith,Mads Melbye,Mads Melbye,Mads Melbye,Bo Jacobsson,Louis J. Muglia +48 more
TL;DR: It is found that variants at the EBF1, EEFSEC, AGTR2, WNT4, ADCY5, and RAP2C loci were associated with gestational duration and variants atThe EBF 1, EE FSEC, and AG TR2 loci with preterm birth.
Journal ArticleDOI
Impaired synaptic plasticity and cAMP response element-binding protein activation in Ca2+/calmodulin-dependent protein kinase type IV/Gr-deficient mice.
Nga Ho,Jason Liauw,Frank Blaeser,Feng Wei,Silva H. Hanissian,Lisa M. Muglia,David F. Wozniak,A. Nardi,Kara L. Arvin,David M. Holtzman,David J. Linden,Min Zhuo,Louis J. Muglia,Talal A. Chatila +13 more
TL;DR: An important role for CaMKIV/Gr in Ca2+-regulated neuronal gene transcription and synaptic plasticity is supported and the contribution of other signaling pathways may spare spatial memory of CaMK IV/Gr-deficient mice are suggested.
Journal ArticleDOI
Genetic Elimination of Behavioral Sensitization in Mice Lacking Calmodulin-Stimulated Adenylyl Cyclases
Feng Wei,Chang Shen Qiu,Susan J. Kim,Lisa M. Muglia,James W. Maas,Victor V. Pineda,Haiming Xu,Zhou-Feng Chen,Daniel R. Storm,Louis J. Muglia,Min Zhuo +10 more
TL;DR: It is shown that wild-type, AC1, AC8, or AC1&8 double knockout mice were indistinguishable in tests of acute pain, whereas behavioral responses to peripheral injection of two inflammatory stimuli, formalin and complete Freund's adjuvant, were reduced or abolished in AC1 &8 DKO mice.
Journal ArticleDOI
Altered Stress-Induced Anxiety in Adenylyl Cyclase Type VIII-Deficient Mice
Michele L. Schaefer,Scott T. Wong,David F. Wozniak,Lisa M. Muglia,Jason Liauw,Min Zhuo,A. Nardi,Richard E. Hartman,Sherri K. Vogt,Christina Luedke,Daniel R. Storm,Louis J. Muglia +11 more
TL;DR: A novel role for AC8 in the modulation of anxiety is demonstrated as a compromise in calcium-stimulated AC activity in the hippocampus, hypothalamus, thalamus, and brainstem and the behavioral consequences of AC8 deficiency are evaluated.
Journal ArticleDOI
T-cell glucocorticoid receptor is required to suppress COX-2-mediated lethal immune activation.
Judson A. Brewer,Bernard Khor,Sherri K. Vogt,Lisa M. Muglia,Hideji Fujiwara,Karen E Haegele,Barry P. Sleckman,Louis J. Muglia +7 more
TL;DR: It is indicated that glucocorticoid receptor suppression of COX-2 is crucial for curtailing lethal immune activation, and new therapeutic approaches for regulation of T-cell-mediated inflammatory diseases are suggested.