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Maria Teresa Sánchez-Aparicio

Researcher at Icahn School of Medicine at Mount Sinai

Publications -  26
Citations -  1807

Maria Teresa Sánchez-Aparicio is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Interferon & Innate immune system. The author has an hindex of 17, co-authored 24 publications receiving 1296 citations. Previous affiliations of Maria Teresa Sánchez-Aparicio include Emerging Pathogens Institute & Spanish National Research Council.

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Dengue virus NS2B protein targets cGAS for degradation and prevents mitochondrial DNA sensing during infection.

TL;DR: It is reported that the DENV NS2B protease cofactor targets the DNA sensor cyclic GMP-AMP synthase for lysosomal degradation to avoid the detection of mitochondrial DNA during infection and results in the inhibition of type I interferon production in the infected cell.
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The E3-Ligase TRIM Family of Proteins Regulates Signaling Pathways Triggered by Innate Immune Pattern-Recognition Receptors

TL;DR: It is demonstrated that roughly half of the 75 TRIM-family members enhanced the innate immune response and that they do this at multiple levels in signaling pathways, which supports the notion that this family of proteins evolved as a component of innate immunity.
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Hijacking of RIG-I Signaling Proteins into Virus-Induced Cytoplasmic Structures Correlates with the Inhibition of Type I Interferon Responses

TL;DR: The data suggest that sequestration of RIG-I signaling molecules into endosome-like structures may be the mechanism used by SFTSV to inhibit IFN responses and point toward a novel mechanism for the suppression ofIFN responses.
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MERS-CoV 4b protein interferes with the NF-κB-dependent innate immune response during infection.

TL;DR: It is found that MERS-CoV 4b was required to prevent a robust NF-κB dependent response during infection, and a model where 4b outcompetes NF-α4 for KPNA4 binding and translocation into the nucleus as a mechanism of interference with the NF-σκB-mediated innate immune response is proposed.