M
Mark A. Eckert
Researcher at Medical University of South Carolina
Publications - 155
Citations - 12085
Mark A. Eckert is an academic researcher from Medical University of South Carolina. The author has contributed to research in topics: Dyslexia & Reading disability. The author has an hindex of 56, co-authored 144 publications receiving 10295 citations. Previous affiliations of Mark A. Eckert include University of Lethbridge & University of Chicago.
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Journal ArticleDOI
White Matter Development During Childhood and Adolescence: A Cross-sectional Diffusion Tensor Imaging Study
Naama Barnea-Goraly,Vinod Menon,Mark A. Eckert,Leanne Tamm,Roland Bammer,Asya Karchemskiy,Christopher Dant,Allan L. Reiss +7 more
TL;DR: It is demonstrated that during childhood and adolescence, white matter anisotropy changes in brain regions that are important for attention, motor skills, cognitive ability, and memory are changed.
Journal ArticleDOI
Hearing impairment and cognitive energy: the Framework for Understanding Effortful Listening (FUEL)
M. Kathleen Pichora-Fuller,Sophia E. Kramer,Mark A. Eckert,Brent Edwards,Benjamin W. Y. Hornsby,Larry E. Humes,Ulrike Lemke,Thomas Lunner,Mohan Matthen,Carol L. Mackersie,Graham Naylor,Natalie A. Phillips,Michael Richter,Mary Rudner,Mitchell S. Sommers,Kelly L. Tremblay,Arthur Wingfield +16 more
TL;DR: This work adapted Kahneman's seminal (1973) Capacity Model of Attention to listening and proposed a heuristically useful Framework for Understanding Effortful Listening (FUEL), which incorporates the well-known relationship between cognitive demand and the supply of cognitive capacity that is the foundation of cognitive theories of attention.
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Developmental Changes in Mental Arithmetic: Evidence for Increased Functional Specialization in the Left Inferior Parietal Cortex
TL;DR: Evidence is provided for a process of increased functional specialization of the left inferior parietal cortex in mental arithmetic, a process that is accompanied by decreased dependence on memory and attentional resources with development.
Journal ArticleDOI
m 6 A mRNA methylation regulates AKT activity to promote the proliferation and tumorigenicity of endometrial cancer
Jun Liu,Jun Liu,Mark A. Eckert,Bryan T. Harada,Bryan T. Harada,Song-Mei Liu,Zhike Lu,Zhike Lu,Kangkang Yu,Kangkang Yu,Kangkang Yu,Samantha M. Tienda,Agnieszka Chryplewicz,Allen Zhu,Allen Zhu,Ying Yang,Jing-Tao Huang,Shao-Min Chen,Zhi-Gao Xu,Xiao-Hua Leng,Xue-Chen Yu,Jie Cao,Zezhou Zhang,Jianzhao Liu,Ernst Lengyel,Chuan He,Chuan He +26 more
TL;DR: Investigation of human endometrial cancer in which a hotspot R298P mutation is present in a key component of the methyltransferase complex reveals reduced m6A mRNA methylation as an oncogenic mechanism in endometricrial cancer and identifies m 6A methylationAs a regulator of AKT signalling.
Journal ArticleDOI
Twist1-Induced Invadopodia Formation Promotes Tumor Metastasis
Mark A. Eckert,Thinzar M. Lwin,Andrew T. Chang,Jihoon Kim,Etienne Danis,Lucila Ohno-Machado,Jing Yang +6 more
TL;DR: It is reported that Twist1 is capable of promoting the formation of invadopodia, specialized membrane protrusions for extracellular matrix degradation, which is a key function of Twist1 in promoting tumor metastasis.