M
Masanobu Kano
Researcher at University of Tokyo
Publications - 285
Citations - 26199
Masanobu Kano is an academic researcher from University of Tokyo. The author has contributed to research in topics: Cerebellum & Purkinje cell. The author has an hindex of 85, co-authored 273 publications receiving 23858 citations. Previous affiliations of Masanobu Kano include Max Planck Society & Osaka University.
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Journal ArticleDOI
Endocannabinoid-Mediated Control of Synaptic Transmission
TL;DR: This review aims to integrate the current understanding of functions of the endocannabinoid system, especially focusing on the control of synaptic transmission in the brain, and summarizes recent electrophysiological studies carried out on synapses of various brain regions and discusses how synaptic transmission is regulated by endoc cannabinoidoid signaling.
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Deficient cerebellar long-term depression and impaired motor learning in mGluR1 mutant mice.
Atsu Aiba,Masanobu Kano,Chong Chen,Chong Chen,Mark E. Stanton,Gregory D. Fox,Karl Herrup,Theresa A. Zwingman,Susumu Tonegawa,Susumu Tonegawa +9 more
TL;DR: It is concluded that mGLUR1 is required for the induction of LTD and that the ataxic behavior and impaired eyeblink conditioning of the mGluR1 mutant mice are primarily due to deficient LTD.
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Endogenous cannabinoids mediate retrograde signals from depolarized postsynaptic neurons to presynaptic terminals.
TL;DR: The results reveal that endogenous cannabinoids mediate retrograde signals from depolarized postsynaptic neurons to presynaptic terminals to cause the reduction of transmitter release.
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Near-infrared deep brain stimulation via upconversion nanoparticle–mediated optogenetics
Shuo Chen,Adam Z. Weitemier,Xiao Zeng,Linmeng He,Xiyu Wang,Yanqiu Tao,Arthur J Huang,Yuki Hashimotodani,Masanobu Kano,Hirohide Iwasaki,Laxmi Kumar Parajuli,Shigeo Okabe,Daniel Boon Loong Teh,Angelo H. All,Iku Tsutsui-Kimura,Kenji F. Tanaka,Xiaogang Liu,Xiaogang Liu,Thomas J. McHugh,Thomas J. McHugh +19 more
TL;DR: It is demonstrated that molecularly tailored UCNPs can serve as optogenetic actuators of transcranial NIR light to stimulate deep brain neurons and evoked dopamine release from genetically tagged neurons in the ventral tegmental area and triggered memory recall.
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Presynaptic Inhibition Caused by Retrograde Signal from Metabotropic Glutamate to Cannabinoid Receptors
TL;DR: A signaling mechanism whereby activation of postsynaptic mGluR retrogradely influences presynaptic functions via endocannabinoid system is highlighted, which can be initiated by two distinct stimuli.