K
Klaus Peter Wandinger
Researcher at University of Hamburg
Publications - 19
Citations - 4123
Klaus Peter Wandinger is an academic researcher from University of Hamburg. The author has contributed to research in topics: T cell & Neuromyelitis optica. The author has an hindex of 13, co-authored 19 publications receiving 3005 citations. Previous affiliations of Klaus Peter Wandinger include National Institutes of Health.
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Journal ArticleDOI
A clinical approach to diagnosis of autoimmune encephalitis
Francesc Graus,Maarten J. Titulaer,Ramani Balu,Susanne M. Benseler,Christian G. Bien,Tania Cellucci,Irene Cortese,Russell C. Dale,Jeffrey M. Gelfand,Michael D. Geschwind,Carol A. Glaser,Jérôme Honnorat,Romana Höftberger,Takahiro Iizuka,Sarosh R. Irani,Eric Lancaster,Frank Leypoldt,Harald Prüss,Alexander Rae-Grant,Markus Reindl,Myrna R. Rosenfeld,Kevin Rostasy,Albert Saiz,Arun Venkatesan,Angela Vincent,Klaus Peter Wandinger,Patrick Waters,J. Dalmau,J. Dalmau +28 more
TL;DR: Through logical differential diagnosis, levels of evidence for autoimmune encephalitis (possible, probable, or definite) are achieved, which can lead to prompt immunotherapy.
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Contrasting disease patterns in seropositive and seronegative neuromyelitis optica: A multicentre study of 175 patients
Sven Jarius,Klemens Ruprecht,Brigitte Wildemann,Tania Kuempfel,Marius Ringelstein,Christian Geis,Ingo Kleiter,Ingo Kleiter,Christoph Kleinschnitz,Achim Berthele,Johannes Brettschneider,Kerstin Hellwig,Bernhard Hemmer,Ralf A. Linker,Ralf A. Linker,Florian Lauda,Christoph Mayer,Hayrettin Tumani,Arthur Melms,Corinna Trebst,Martin Stangel,Martin Marziniak,Frank Hoffmann,Sven Schippling,Jürgen H. Faiss,Oliver Neuhaus,Barbara Ettrich,Christian Zentner,Kersten Guthke,Ulrich Hofstadt-van Oy,Reinhard Reuss,Hannah L. Pellkofer,Ulf Ziemann,Peter Kern,Klaus Peter Wandinger,Florian Then Bergh,Tobias Boettcher,Stefan Langel,Martin Liebetrau,Paulus S. Rommer,Sabine Niehaus,Christoph Münch,Alexander Winkelmann,Uwe K Zettl U,Imke Metz,Christian Veauthier,Jörn P Sieb,C. Wilke,Hans Hartung,Orhan Aktas,Friedemann Paul +50 more
TL;DR: This study provides an overview of the clinical and paraclinical features of NMOSD in Caucasians and demonstrates a number of distinct disease characteristics in seropositive and seronegative patients.
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Low-Avidity CD4 + T Cell Responses to SARS-CoV-2 in Unexposed Individuals and Humans with Severe COVID-19.
Petra Bacher,Elisa Rosati,Daniela Esser,Gabriela Rios Martini,Carina Saggau,Esther Schiminsky,Justina Dargvainiene,Ina Schröder,Imke Wieters,Yascha Khodamoradi,Fabian J. Eberhardt,Maria J G T Vehreschild,Holger Neb,Michael Sonntagbauer,Claudio Conrad,Florian Tran,Philip Rosenstiel,Robert Markewitz,Klaus Peter Wandinger,Max Augustin,Jan Rybniker,Matthias Kochanek,Frank Leypoldt,Oliver A. Cornely,Philipp Koehler,Andre Franke,Alexander Scheffold +26 more
TL;DR: These findings identify low avidity CD4+T cell responses as a hallmark of severe COVID-19, and argue against a protective role for CCCoV reactive T cells in SARS-CoV-2 infection.
Journal ArticleDOI
Complex immunomodulatory effects of interferon‐β in multiple sclerosis include the upregulation of T helper 1‐associated marker genes
Klaus Peter Wandinger,Claus Steffen Stürzebecher,Bibiana Bielekova,Greg Detore,Andreas Rosenwald,Louis M. Staudt,Henry F. McFarland,Roland Martin +7 more
TL;DR: The combination of cDNA microarray and quantitative polymerase chain reaction will expand the knowledge of the immunological effects of such pleiotropic agents as IFN‐β, may provide a key to why certain patients fail to respond, and eventually influence the view of the disease pathogenesis.
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Frequency and syndrome specificity of antibodies to aquaporin-4 in neurological patients with rheumatic disorders
Sven Jarius,Christian Jacobi,Jérôme De Seze,Hélène Zéphir,Friedemann Paul,Diego Franciotta,Paulus S. Rommer,Simone Mader,Ingo Kleiter,Markus Reindl,Gulsen Akman-Demir,Thomas Seifert-Held,Wolfgang Kristoferitsch,Arthur Melms,Klaus Peter Wandinger,Brigitte Wildemann +15 more
TL;DR: The high syndrome specificity of the antibody for neuromyelitis optica spectrum disorders (NMOSDs) in patients with CTD supports the concept of AQP4-Ab being involved in the pathogenesis of these neurological conditions, and argues against AQP 4-Ab simply being part of the polyclonal B cell activation generally associated with rheumatic diseases.