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Pasqua Veronico

Researcher at University of Bari

Publications -  21
Citations -  1510

Pasqua Veronico is an academic researcher from University of Bari. The author has contributed to research in topics: Meloidogyne incognita & Gene. The author has an hindex of 13, co-authored 18 publications receiving 1285 citations.

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Genome sequence of the metazoan plant-parasitic nematode Meloidogyne incognita

Pierre Abad, +76 more
- 27 Jul 2008 - 
TL;DR: The draft genome sequence of the root-knot nematode Meloidogyne incognita, a biotrophic parasite of many crops, is reported, providing insights into the adaptations required by metazoans to successfully parasitize immunocompetent plants, and open the way for discovering new antiparasitic strategies.
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Nematode chitin synthases: gene structure, expression and function in Caenorhabditis elegans and the plant parasitic nematode Meloidogyne artiellia.

TL;DR: A full-length genomic sequence of a chitin synthase orthologue from the plant parasitic nematode Meloidogyne artiellia is isolated and shows an expression pattern that is consistent with a role for the protein in production of the eggshell.
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A novel lipoxygenase in pea roots. Its function in wounding and biotic stress

TL;DR: In situ hybridization revealed that LOXN2 message was widespread in the cortex and endodermis of healthy roots, but specifically localized at high level in the cells bordering the nematode-induced syncytia of infected roots, suggesting LOXn2 involvement in late mechanisms of host resistance.
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Analysis of class III peroxidase genes expressed in roots of resistant and susceptible wheat lines infected by Heterodera avenae.

TL;DR: Data seem to suggest that wheat apoplastic peroxidases, because of their different expression in quantity and timing, play different roles in the plant response to nematode infection.
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ROS and NO production in compatible and incompatible tomato- Meloidogyne incognita interactions

TL;DR: A functional overlap of both NO and ROS is envisaged in tomato defence response to nematode invasion, NO and H2O2 cooperating in triggering hypersensitive cell death, and which may influence the host cellular metabolism.