P
Peter R. Dodd
Researcher at University of Queensland
Publications - 220
Citations - 12265
Peter R. Dodd is an academic researcher from University of Queensland. The author has contributed to research in topics: Glutamate receptor & Human brain. The author has an hindex of 52, co-authored 219 publications receiving 11627 citations. Previous affiliations of Peter R. Dodd include University of New South Wales & Imperial College London.
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Journal ArticleDOI
Association of missense and 5′-splice-site mutations in tau with the inherited dementia FTDP-17
Mike Hutton,Corinne Lendon,Patrizia Rizzu,Matt Baker,Susanne Froelich,Susanne Froelich,Henry Houlden,Stuart Pickering-Brown,Sumitra Chakraverty,Adrian M. Isaacs,Andrew Grover,J. Hackett,Jennifer Adamson,Sarah Lincoln,Dennis W. Dickson,Peter Davies,Ronald C. Petersen,M. Stevens,E. De Graaff,E. Wauters,J. Van Baren,M. Hillebrand,Marijke Joosse,J. M. Kwon,Petra Nowotny,Lien Kuei Che,Joanne Norton,John C. Morris,L. A. Reed,John Q. Trojanowski,Hans Basun,Lars Lannfelt,M. Neystat,Stanley Fahn,Frances Dark,Tony Tannenberg,Peter R. Dodd,Nicholas K. Hayward,John B.J. Kwok,Peter R. Schofield,Athena Andreadis,Julie S. Snowden,David Craufurd,David Neary,F. Owen,Ben A. Oostra,John Hardy,Alison Goate,J. C. van Swieten,David M. A. Mann,Timothy Lynch,Peter Heutink +51 more
TL;DR: In this paper, the authors sequenced tau in FTDP-17 families and identified three missense mutations (G272V, P301L and R406W) and three mutations in the 5' splice site of exon in
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Glutamate-mediated excitotoxicity and neurodegeneration in Alzheimer's disease.
TL;DR: Mechanisms for the involvement of the NMDA receptor complex and its interaction with polyamines in the pathogenesis of AD are discussed andNMDA receptor antagonists have potential for the therapeutic amelioration of AD.
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A Rapid Method for Preparing Synaptosomes - Comparison, with Alternative Procedures
TL;DR: The new preparation method has been found to be of value in metabolic studies of synaptosomes prepared from human post-mortem brain and compared favourably with alternative methods as judged by indices of metabolic and functional performance.
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Gene expression in human alcoholism: microarray analysis of frontal cortex.
Joanne Marie Lewohl,Joanne Marie Lewohl,Long Wang,Michael F. Miles,Li Zhang,Peter R. Dodd,R. Adron Harris +6 more
TL;DR: These gene expression changes suggest a mechanism for the loss of cerebral white matter in alcoholics as well as alterations that may lead to the neurotoxic actions of ethanol.
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Patterns of gene expression are altered in the frontal and motor cortices of human alcoholics.
TL;DR: Examination of expression patterns in frontal and motor cortices of three groups of chronic alcoholic and matched control cases suggests that multiple pathways may be important for neuropathology and altered neuronal function observed in alcoholism.