R
Raghu Kalluri
Researcher at University of Texas MD Anderson Cancer Center
Publications - 325
Citations - 89851
Raghu Kalluri is an academic researcher from University of Texas MD Anderson Cancer Center. The author has contributed to research in topics: Cancer & Angiogenesis. The author has an hindex of 115, co-authored 306 publications receiving 71127 citations. Previous affiliations of Raghu Kalluri include Beth Israel Deaconess Medical Center & Baylor College of Medicine.
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Journal ArticleDOI
Proteinuria with and without Renal Glomerular Podocyte Effacement
TL;DR: It was demonstrated that proteinuria can be observed without podocyte foot process effacement in three different mouse models and the supporting evidence from several human studies demonstrate that severe defects in either the glomerular basement membrane or the glomersular endothelium can lead to proteinuria without foot process Effacement.
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Physiology of the renal interstitium
Michael Zeisberg,Raghu Kalluri +1 more
TL;DR: The functional role of renal interstitium is reviewed, with identification of interstitial renin- and erythropoietin-producing cells, the most prominent endocrine functions of the kidney have now been attributed to the renal interStitium.
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Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer.
Pingping Hou,Avnish Kapoor,Qiang Zhang,Jiexi Li,Chang-Jiun Wu,Jun Li,Zhengdao Lan,Ming Tang,Xingdi Ma,Jeffrey J. Ackroyd,Raghu Kalluri,Jianhua Zhang,Shan Jiang,Denise J. Spring,Y. Alan Wang,Ronald A. DePinho +15 more
TL;DR: In this article, the authors explored mechanisms involving the tumor microenvironment (TME) as a potential basis for resistance to targeting KRAS*, using the inducible KrasG12D p53 null (iKPC) PDAC mouse model.
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Cellular and molecular pathways that lead to progression and regression of renal fibrogenesis.
Hirokazu Okada,Raghu Kalluri +1 more
TL;DR: This review highlights recent advances in the understanding of the cellular and molecular events leading to the progression of renal fibrosis.
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Low-dose hydralazine prevents fibrosis in a murine model of acute kidney injury–to–chronic kidney disease progression
Björn Tampe,Ulrike Steinle,Désirée Tampe,Julienne L. Carstens,Peter Korsten,Elisabeth M. Zeisberg,Gerhard A. Müller,Raghu Kalluri,Michael Zeisberg +8 more
TL;DR: A murine model of ischemia-reperfusion injury is used to determine whether aberrant promoter methylation of RASAL1 contributes causally to the switch between physiological regeneration and tubulointerstitial fibrogenesis, a hallmark of AKI-to-CKD progression.