scispace - formally typeset
R

Russell A. Poldrack

Researcher at Stanford University

Publications -  481
Citations -  70423

Russell A. Poldrack is an academic researcher from Stanford University. The author has contributed to research in topics: Cognition & Functional neuroimaging. The author has an hindex of 125, co-authored 452 publications receiving 58695 citations. Previous affiliations of Russell A. Poldrack include University of Illinois at Urbana–Champaign & University of Texas at Austin.

Papers
More filters
Journal ArticleDOI

Differences in the hemodynamic response to event-related motor and visual paradigms as measured by near-infrared spectroscopy

TL;DR: The results demonstrate that rapid, event-related NIRS can provide robust estimates of the hemodynamic response without artifacts due to low-frequency signal components, unlike data from blocked designs, and suggests that this difference may be attributable to different capillary transit times in these cortices.
Journal ArticleDOI

Data sharing in neuroimaging research

TL;DR: The INCF Task Force on Neuroimaging Datasharing, in conjunction with several collaborative groups around the world, has started work on several tools to ease and eventually automate the practice of data sharing, hoped that such tools will allow researchers to easily share raw, processed, and derived neuroimaging data, with appropriate metadata and provenance records, and will improve the reproducibility of neuroim imaging studies.
Journal ArticleDOI

Large-scale analysis of test–retest reliabilities of self-regulation measures

TL;DR: It is found that dependent variables from self-report surveys of self-regulation have high test–retest reliability, while DVs derived from behavioral tasks do not, and it is confirmed that this is due to differences in between-subject variability.
Journal ArticleDOI

Altered Functional Connectivity in Frontal Lobe Circuits Is Associated with Variation in the Autism Risk Gene CNTNAP2

TL;DR: Using functional neuroimaging, a relationship between frontal lobar connectivity and common genetic variants in CNTNAP2 is demonstrated, providing a mechanistic link between specific genetic risk for neurodevelopmental disorders and empirical data implicating dysfunction of long-range connections within the frontal lobe in autism.