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Takashi Saito

Researcher at Nagoya City University

Publications -  1220
Citations -  60014

Takashi Saito is an academic researcher from Nagoya City University. The author has contributed to research in topics: Medicine & MAGIC (telescope). The author has an hindex of 112, co-authored 1041 publications receiving 52937 citations. Previous affiliations of Takashi Saito include Mitsubishi Electric & Nippon Medical School.

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Ruptured left coronary sinus of valsalva aneurysm into the left ventricle.

TL;DR: A 59-year-old Japanese male presented with exertional dyspnea and Histologic examination showed an accumulation of inflammatory cells implying that a possible cause of aneurysmal formation was an infective endocarditis although any organisms could be identified from these specimens.
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Retinal Thickness Changes Over Time in a Murine AD Model APP NL-F/NL-F.

TL;DR: In this article, the authors used spectral domain optical coherence tomography (SD-OCT) to determine the total retinal thickness and retinal nerve fiber layer (RNFL) thickness in an APPNL-F/NL-F mouse model of AD at 6, 9, 12, 15, 17, and 20 months old compared to wild type (WT) animals.
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Episodic tremors representing cortical myoclonus are characteristic in Angelman syndrome due to UBE3A mutations

TL;DR: Semirhythmic myoclonus is common in patients with Angelman syndrome caused by UBE3A mutations, and such myoclonic events are often life disabling, which might explain the low prevalence of absence seizures in this population.
Patent

Registration accuracy measurement mark

TL;DR: In this article, a registration accuracy measurement mark taking into consideration the influence of aberration can be provided, where a first semiconductor element forming member formed in a first layer, a first measurement mark formed by the same manufacturing step as the first semiconducter forming member, and a second measurement mark forming in a second layer above the first layer for measuring registration accuracy between the first and second semiconductor elements forming members.
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Terminal complement pathway activation drives synaptic loss in Alzheimer’s disease models

TL;DR: In this paper , the role of the lytic membrane attack complex (MAC) is investigated in synapse loss in Alzheimer's disease. But the precise mechanisms of complement-mediated synaptic loss remain unclear, and the role role of MAC is unexplored.