T
Takashi Saito
Researcher at Nagoya City University
Publications - 1220
Citations - 60014
Takashi Saito is an academic researcher from Nagoya City University. The author has contributed to research in topics: Medicine & MAGIC (telescope). The author has an hindex of 112, co-authored 1041 publications receiving 52937 citations. Previous affiliations of Takashi Saito include Mitsubishi Electric & Nippon Medical School.
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Journal ArticleDOI
4PBA reduces growth deficiency in osteogenesis imperfecta by enhancing transition of hypertrophic chondrocytes to osteoblasts
Amanda L. Scheiber,Kevin J. Wilkinson,Akiko Suzuki,Motomi Enomoto-Iwamoto,Takashi Saito,Kathryn S.E. Cheah,Masahiro Iwamoto,Sergey Leikin,Satoru Otsuru +8 more
TL;DR: It is demonstrated that HC dysfunction induced by ER disruption plays a critical role in the pathogenesis of OI growth deficiency, which lays the foundation for developing new therapies for OI.
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Molecular Cloning of pTAC12 an Alternative Splicing Product of the CD3γ Chain as a Component of the Pre-T Cell Antigen-Receptor Complex
TL;DR: It is reported that pTAC12 is an alternatively spliced product of the CD3γ chain lacking exon 4 containing the transmembrane region and may play a special role for the transport/expression and assembly of the pre-TCR·CD3 complex as well as the clonotype-independent CD3 complex in immature thymocytes.
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Brain endothelial cells produce amyloid-β from amyloid precursor protein 770 and preferentially secrete the O-glycosylated form
Yuriko Tachida,Masaki Kato,Yoshiki Yamaguchi,Takashi Honda,Yasuhiro Hashimoto,Yoshinao Wada,Takashi Saito,Nobuhisa Iwata,Naoyuki Taniguchi +8 more
TL;DR: The data suggest Aß oligomers induce the c-Abl kinase activation through the EphA4 and this could be connected with synaptic demise.
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trans‐[(Mo6Cl8)(C7H7)4{P(n‐C4H9)3}2] and trans‐[(Mo6Cl8)(C8H5)4{P(n‐C5H11)3}2].2C7H8
TL;DR: The octahedral Mo6 cores of both compounds are on inversion centers and are almost regular as discussed by the authors, and the average interatomic distances in the clusters for (1) are Mo-Mo(edge) = 2.618, Mo⋯Mo(opposite) = 3.13
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Microglia and CD206+ border-associated mouse macrophages maintain their embryonic origin during Alzheimer's disease.
TL;DR: In this article, the authors conducted a comprehensive fate-mapping analysis of murine microglia and border-associated macrophages and their turnover kinetics during Alzheimer's disease (AD) progression.