T
Tilman B. Drüeke
Researcher at French Institute of Health and Medical Research
Publications - 415
Citations - 22817
Tilman B. Drüeke is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Kidney disease & Hyperparathyroidism. The author has an hindex of 68, co-authored 413 publications receiving 21471 citations. Previous affiliations of Tilman B. Drüeke include Versailles Saint-Quentin-en-Yvelines University & University of Paris-Sud.
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Molecular characteristics of uronic-acid-rich protein, a strong inhibitor of calcium oxalate crystallization in vitro.
TL;DR: Uronic-acid-rich protein (UAP) is a new urinary macromolecule which strongly inhibits calcium oxalate crystal formation and exhibits partial structural homology with alpha 1-microglobulin.
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Dietary salt restriction accelerates atherosclerosis in apolipoprotein E-deficient mice.
Ognen Ivanovski,Dorota Szumilak,Thao Nguyen-Khoa,Michele Dechaux,Ziad A. Massy,Olivier Phan,Nadya Mothu,Bernard Lacour,Tilman B. Drüeke,Martin S. Muntzel +9 more
TL;DR: Dietary salt restriction accelerated atherosclerotic lesion formation in apoE(-/-) mice through a mechanism that is probably related to ANG-II formation, and whether these findings are relevant to human cardiovascular disease remains to be evaluated.
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Sclerostin: Just One More Player in Renal Bone Disease?
TL;DR: Renal osteodystrophy (ROD) is one of the three components of chronic kidney disease, including mineral and bone disorder (CKD-MBD) as discussed by the authors.
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Lanthanum carbonate as a first-line phosphate binder: the "cons".
TL;DR: The experience with aluminum hydroxide suggests caution regarding the long‐term use of another metal‐based agent that displays enhanced absorption in the uremic state and progressive tissue accumulation.
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Paricalcitol as compared with calcitriol in patients undergoing hemodialysis
TL;DR: Secondary hyperparathyroidism, a common consequence of chronic kidney disease, results from abnormal regulation of calcium and phosphate homeostasis and three factors come into play: renal phosphate clearance and net calcium balance become inadequate to maintain serum phosphorus and ionized calcium levels within an optimal range.