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Tilman B. Drüeke

Researcher at French Institute of Health and Medical Research

Publications -  415
Citations -  22817

Tilman B. Drüeke is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Kidney disease & Hyperparathyroidism. The author has an hindex of 68, co-authored 413 publications receiving 21471 citations. Previous affiliations of Tilman B. Drüeke include Versailles Saint-Quentin-en-Yvelines University & University of Paris-Sud.

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Abnormal intestinal regulation of calbindin-D9K and calmodulin by dietary calcium in genetic hypertension.

TL;DR: Calbindin-D9K was significantly decreased by the high-calcium diet in both strains at both ages and there was a significant correlation between duodenal cal bindin- D9K and plasma levels of calcitriol.
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Effects of saline loading on jejunal absorption of calcium, sodium, and water, and on parathyroid hormone secretion in the rat.

TL;DR: It is concluded that after extracellular fluid volume expansion, net jejunal calcium absorption is decreased and the decrease parallels that of sodium and water; b-l calcium transport is enhanced to a greater degree by calcium-free Ringer infusion than by a calcium-rich solution.

Effects of cinacalcet on atherosclerotic and nonatherosclerotic cardiovascular events in patients receiving hemodialysis: the EValuation Of Cinacalcet HCl Therapy to Lower CardioVascular Events (EVOLVE) trial

TL;DR: In this paper, the authors defined the frequency of fatal and nonfatal cardiovascular events attributable to atherosclerotic and nonatherosclerosis mechanisms, risk factors for these events, and the effects of cinacalcet, using adjudicated data collected during the EValuation of CINACALcet HCl Therapy to Lower CardioVascular Events (EVOLVE) trial.
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Less arterial stiffness in kidney transplant recipients than chronic kidney disease patients matched for renal function.

TL;DR: Aortic stiffness was significantly less marked in KTRs 1 year post- TransplanTest than in CKDps matched for GFR and other variables, compatible with the view that the pathogenesis of post-transplant cardiovascular disease differs, at least in part, from that of CKD per se.