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Walter Malorni

Researcher at Istituto Superiore di Sanità

Publications -  371
Citations -  39681

Walter Malorni is an academic researcher from Istituto Superiore di Sanità. The author has contributed to research in topics: Apoptosis & Programmed cell death. The author has an hindex of 69, co-authored 363 publications receiving 33990 citations.

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Book ChapterDOI

Analyzing lipid raft dynamics during cell apoptosis.

TL;DR: This chapter focuses mainly on coimmunoprecipitation methods, which represent a useful tool in analyzing raft dynamics during apoptosis, and the combined use of all these different approaches appears to be mandatory for analyzing the role of lipid raft dynamicsDuring apoptosis.
Journal ArticleDOI

Gender-specific features of plasmatic and circulating cell alterations as risk factors in cardiovascular disease.

TL;DR: An overview on current knowledge on sex‐associated cardiovascular determinants is provided with the aim to improve CVD diagnostic and prognostic clinical courses and to develop new and gender‐biased prevention strategies.
Journal ArticleDOI

P-Selectin as a new gender associated biomarker in patients with metabolic syndrome.

TL;DR: Corretti et al. as discussed by the authors proposed a surrogate marker for arterial wall thickness as a surrogate for atherosclerosis in patients with aneurysmal coronary artery disease, which is based on ultrasound assessment of endothelial-dependent flow-mediated vasodilation.
Journal Article

Cellular mechanisms of lymphocyte-mediated lysis of tumor cells.

TL;DR: A growing body of evidence has recently supplied proof that target cells may play an active role in their own lysis supporting the challenging opinion of an induced suicide mechanism.
Journal ArticleDOI

Effect of lonidamine on the aerobic glycolysis of normal and phytohemagglutinin-stimulated human peripheral blood lymphocytes

TL;DR: It is suggested that the inhibition of mitochondria-bound hexokinase might be ascribed to marked changes in membrane conformation that affect the activity of membrane-associated enzymes, rather than to a direct effect of the enzyme itself.