scispace - formally typeset
R

Roberta Misasi

Researcher at Sapienza University of Rome

Publications -  135
Citations -  4827

Roberta Misasi is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Lipid raft & Antiphospholipid syndrome. The author has an hindex of 35, co-authored 123 publications receiving 3504 citations. Previous affiliations of Roberta Misasi include Istituto Superiore di Sanità & University of California, San Diego.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Anti-β2-glycoprotein I antibodies induce monocyte release of tumor necrosis factor α and tissue factor by signal transduction pathways involving lipid rafts

TL;DR: New insight is provided into the pathogenesis of APS and anti-β2GPI antibodies react with their target antigen, likely in association with annexin A2 and TLR-4, in lipid rafts in the monocyte plasma membrane, improving the knowledge of valuable therapeutic targets.
Journal ArticleDOI

Identification of a neurotrophic sequence in erythropoietin.

TL;DR: Findings indicate that neural cells and not hematological cells respond to a peptide sequence within erythropoietin and suggests that Epo may have separate domains for neurotrophic and hematotrophic function.
Journal ArticleDOI

Evidence for the involvement of lipid rafts localized at the ER-mitochondria associated membranes in autophagosome formation.

TL;DR: It is hypothesized that MAM raft-like microdomains could be pivotal in the initial organelle scrambling activity that finally leads to the formation of autophagosome.
Journal ArticleDOI

Lipid microdomains contribute to apoptosis-associated modifications of mitochondria in T cells

TL;DR: Mitochondrion appears as a subcompartmentalized organelle, in which microdomains may act as controllers of their apoptogenic programs, including fission-associated morphogenetic changes, megapore formation and function.