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Journal ArticleDOI

Calcium transport in small intestine during pregnancy and lactation.

TLDR
Calcium transport in the intestine increases late in pregnancy, peaks during lactation, and then falls to control values by 3 wk postweaning in both vitamin D-replete and D-deficient animals, suggesting that some factor(s) independent of vitamin D is stimulating intestinal calcium transport during the reproductive cycle.
Abstract
The factors involved in calcium homeostasis during the mammalian reproductive cycle and specifically in the control of active calcium transport in the intestine have not been thoroughly investigate...

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Citations
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Maternal-fetal calcium and bone metabolism during pregnancy, puerperium, and lactation.

TL;DR: Maternal adaptive goals during pregnancy and during lactation, andfetal response to maternal hyper- or hypoparathy-roidism, and Integrated fetal calcium homeostasis, are studied.
Journal ArticleDOI

The physiological costs of reproduction in small mammals

TL;DR: Life-history trade-offs between components of fitness arise because reproduction entails both gains and costs, and knowledge of these physiological costs is currently at best described as rudimentary.
Journal ArticleDOI

A longitudinal study of calcium homeostasis during human pregnancy and lactation and after resumption of menses.

TL;DR: Fetal calcium demand was met by increased maternal intestinal absorption; early breast-milk calcium was provided by maternal renal calcium conservation and loss of spinal trabecular bone, a loss that was recovered postmenses.
Journal ArticleDOI

Vitamin D in pregnancy and lactation: maternal, fetal, and neonatal outcomes from human and animal studies

TL;DR: Dosing recommendations for women during pregnancy and lactation might be best directed toward ensuring that the neonate is vitamin D-sufficient and that this sufficiency is maintained during infancy and beyond.
Journal ArticleDOI

Maternal Mineral and Bone Metabolism During Pregnancy, Lactation, and Post-Weaning Recovery

TL;DR: This review addresses the current knowledge regarding maternal adaptations in mineral and skeletal homeostasis that occur during pregnancy, lactation, and post-weaning recovery and the impacts that these adaptations have on biochemical and hormonal parameters of mineralHomeostasis.
References
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Journal ArticleDOI

Control of 25-hydroxycholecalciferol metabolism by parathyroid glands.

TL;DR: It appears that the parathyroid hormone serves as a tropin for production of 1,25-dihydroxycholecalciferol, the hormonal form of vitamin D responsible for calcium mobilization from intestinal contents and bone.
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A sensitive, precise, and convenient method for determination of 1,25-dihydroxyvitamin D in human plasma☆

TL;DR: With this method, rachitic chick plasma, plasma from anephric patients, and plasma from patients suffering severe endstage renal failure show no detectable 1,25-dihydroxyvitamin D, while normal human values have been found to be 29 ± 2 pg/ml.
Journal ArticleDOI

Regulation of 25-Hydroxycholecalciferol-1-Hydroxylase Activity in Kidney by Parathyroid Hormone

TL;DR: Parathyroid hormone, secreted in response to decreasing blood calcium concentration, stimulates formation of the kidney hormone, 1,25-dihydroxy-cholecalciferol.
Journal ArticleDOI

Serum prolactin levels in rats during different reproductive states.

TL;DR: Rat serum prolactin as determined by radioimmunoassay during the estrous cycle was found to be highest during estrus (68.5 ±7.4 ng/ml) and lowest in diestrus (27.6 ± 5.0 ng/ML), and there were no cyclic changes in serum prolACTin levels in mature male rats.
Journal ArticleDOI

Calcium Metabolism in Normal Human Pregnancy

TL;DR: Intestinal calcium absorption was twice usual normal levels from the earliest period studied and remained high throughout pregnancy, and there was a 20% increase in miscible calcium pool by term, and an approximate doubling of both pool turnover and bone mineral accretion rates.
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