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Blazenka Soldo

Researcher at University of Lausanne

Publications -  20
Citations -  4774

Blazenka Soldo is an academic researcher from University of Lausanne. The author has contributed to research in topics: Bacillus subtilis & Gene. The author has an hindex of 17, co-authored 20 publications receiving 4636 citations.

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The complete genome sequence of the Gram-positive bacterium Bacillus subtilis

F. Kunst, +154 more
- 20 Nov 1997 - 
TL;DR: Bacillus subtilis is the best-characterized member of the Gram-positive bacteria, indicating that bacteriophage infection has played an important evolutionary role in horizontal gene transfer, in particular in the propagation of bacterial pathogenesis.
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Sequencing and analysis of the Bacillus subtilis lytRABC divergon: a regulatory unit encompassing the structural genes of the N-acetylmuramoyl-L-alanine amidase and its modifier.

TL;DR: The regulatory unit of Bacillus subtilis strain 168 encompassing the structural genes of the N-acetylmuramoyl-L-alanine amidase and of its modifier has been sequenced, and found to be a divergon consisting of divergently transcribed operons lytABC and lytR.
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tagO is involved in the synthesis of all anionic cell-wall polymers in Bacillus subtilis 168.

TL;DR: The authors discuss the possibility that TagO may represent a pivotal element in the multi-enzyme complexes responsible for the synthesis of anionic cell-wall polymers, and that it may play one of the key roles in balanced cell growth.
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Nucleotide sequence of the Bacillus subtilis temperate bacteriophage SPβc2

TL;DR: The Bacillus subtilis 168 chromosomal region extending from 184 degrees to 195 degrees, corresponding to prophage SPbeta, has been completely sequenced using DNA of the thermoinducible SPbetac2 mutant, and this 134416 bp segment comprises 187 putative ORFs which, according to their orientation, were grouped into three clusters.
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Bacillus subtilis α-phosphoglucomutase is required for normal cell morphology and biofilm formation

TL;DR: The deficiency in biofilm formation in gtaC and gtaE mutants may be attributed to an inability to synthesize UDP-glucose, an important intermediate in a number of cell envelope biosynthetic processes.