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Chizu Ishii

Researcher at Saitama University

Publications -  18
Citations -  1590

Chizu Ishii is an academic researcher from Saitama University. The author has contributed to research in topics: Neurospora crassa & Mutant. The author has an hindex of 13, co-authored 18 publications receiving 1515 citations. Previous affiliations of Chizu Ishii include Tohoku University.

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Lessons from the Genome Sequence of Neurospora crassa: Tracing the Path from Genomic Blueprint to Multicellular Organism

TL;DR: An analysis of over 1,100 of the ∼10,000 predicted proteins encoded by the genome sequence of the filamentous fungus Neurospora crassa reveals potential new targets for antifungals as well as loci implicated in human and plant physiology and disease.
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Highly efficient gene replacements in Neurospora strains deficient for nonhomologous end-joining.

TL;DR: Results indicate that KU disruption strains are efficient recipients for gene targeting, and identify and disrupted Neurospora crassa genes homologous to human KU70 and KU80, which encode proteins that function in nonhomologous end-joining of double-stranded DNA breaks.
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A eukaryotic gene encoding an endonuclease that specifically repairs DNA damaged by ultraviolet light.

TL;DR: A new eukaryotic DNA repair gene is isolated from N.crassa by its ability to complement UV‐sensitive Escherichia coli cells and confers UV‐resistance on various DNA repair‐deficient mutants of Saccharomyces cerevisiae and a human xeroderma pigmentosum cell line.
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Isolation and characterization of MMS-sensitive mutants of Neurosporacrassa

TL;DR: At least two new loci involved in the repair of MMS damage have been identified and the possibility that some of these new mutants are in new repair pathways is suggested.
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Characterization of a Neurospora crassa photolyase-deficient mutant generated by repeat induced point mutation of the phr gene.

TL;DR: The results show that the phr mutation has no influence on dark repair, and N. crassa photolyase is not a blue light receptor in the signal transduction that induces carotenoid biosynthesis.