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Erik Ingelsson

Researcher at Stanford University

Publications -  546
Citations -  99427

Erik Ingelsson is an academic researcher from Stanford University. The author has contributed to research in topics: Genome-wide association study & Population. The author has an hindex of 124, co-authored 538 publications receiving 85407 citations. Previous affiliations of Erik Ingelsson include Karolinska Institutet & Cardiovascular Institute of the South.

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Genetic factors may play a prominent role in the development of coronary heart disease dependent on important environmental factors

TL;DR: The aim of the study was to examine whether various lifestyle factors modify genetic influences on coronary heart disease (CHD) and to investigate the role of diet, exercise, and diet in this disease.
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Urinary Albumin, Sodium, and Potassium and Cardiovascular Outcomes in the UK Biobank: Observational and Mendelian Randomization Analyses.

TL;DR: A positive association between UNa/UK and albumin with blood pressure (BP), as well as with adiposity-related measures is found, and the finding of a bidirectional causal association between albumin and T2D reflects the well-known nephropathy in T1D.
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Influence of persistent organic pollutants on the complement system in a population-based human sample.

TL;DR: It was shown that POPs, mainly PCB-126, were associated with levels of complement system markers indicating that the association of these toxic compounds with downstream disease could be mediated by activation of immune system.
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Adiponectin and cardiac geometry and function in elderly: results from two community-based cohort studies

TL;DR: Serum adiponectin concentrations were associated with ejection fraction in men, and these associations were partially attenuated by NT-proBNP, implying that adiponECTin may be associated with systolic function through pathways that involve natriuretic peptides.
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The role of obesity-related genetic loci in insulin sensitivity.

TL;DR: Despite rapid advancements and many new diabetes susceptibility loci found in the past few years, few genetic variants associated with insulin sensitivity have been described, potentially attaining high insulin sensitivity as discussed by the authors.