E
Erik Ingelsson
Researcher at Stanford University
Publications - 546
Citations - 99427
Erik Ingelsson is an academic researcher from Stanford University. The author has contributed to research in topics: Genome-wide association study & Population. The author has an hindex of 124, co-authored 538 publications receiving 85407 citations. Previous affiliations of Erik Ingelsson include Karolinska Institutet & Cardiovascular Institute of the South.
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Journal ArticleDOI
Dose–Response Relationship of Total and Leisure Time Physical Activity to Risk of Heart Failure : a prospective cohort study
Kasper Andersen,Daniela Mariosa,Hans-Olov Adami,Claes Held,Erik Ingelsson,Ylva Trolle Lagerros,Olof Nyrén,Weimin Ye,Rino Bellocco,Johan Sundström +9 more
TL;DR: In this paper, the authors investigated nonlinear associations of total and leisure time physical activeness with risk of heart failure, and found that the relationship between levels of physical activity and risk of coronary heart failure was not well understood.
Journal ArticleDOI
Large-Scale Phenome-Wide Association Study of PCSK9 Variants Demonstrates Protection Against Ischemic Stroke.
Abhiram Rao,Daniel Lindholm,Manuel A. Rivas,Joshua W. Knowles,Stephen B. Montgomery,Erik Ingelsson +5 more
TL;DR: This result represents the first genetic evidence in a large cohort for the protective effect of PCSK9 inhibition on ischemic stroke and corroborates exploratory evidence from clinical trials.
Journal ArticleDOI
Genetic determinants of mortality. Can findings from genome-wide association studies explain variation in human mortality?
Andrea Ganna,Fernando Rivadeneira,Fernando Rivadeneira,Albert Hofman,André G. Uitterlinden,André G. Uitterlinden,Patrik K. E. Magnusson,Nancy L. Pedersen,Erik Ingelsson,Henning Tiemeier +9 more
TL;DR: The findings are compatible with the view of mortality as a complex and highly polygenetic trait, not easily explainable by common genetic variants related to diseases and physiological traits.
Journal ArticleDOI
Association Between Circulating Endostatin, Hypertension Duration, and Hypertensive Target-Organ Damage
Axel C. Carlsson,Toralph Ruge,Johan Sundström,Erik Ingelsson,Anders Larsson,Lars Lind,Johan Ärnlöv +6 more
TL;DR: Circulating endostatin is associated with the duration of hypertension, and vascular, myocardial, and renal indices of hypertensive target-organ damage.
Journal ArticleDOI
Loss of cardioprotective effects at the ADAMTS7 locus as a result of gene-smoking interactions
Danish Saleheen,Wei Zhao,Robin Young,Christopher P. Nelson,WeangKee Ho,Jane F. Ferguson,Jane F. Ferguson,Asif Rasheed,Kristy Ou,Sylvia T. Nurnberg,Sylvia T. Nurnberg,Robert C. Bauer,Robert C. Bauer,Anuj Goel,Ron Do,Ron Do,Alexandre F.R. Stewart,Alexandre F.R. Stewart,Jaana Hartiala,Jaana Hartiala,Weihua Zhang,Gudmar Thorleifsson,Rona J. Strawbridge,Rona J. Strawbridge,Juha Sinisalo,Stavroula Kanoni,Stavroula Kanoni,Sanaz Sedaghat,Eirini Marouli,Eirini Marouli,K. Kristiansson,K. Kristiansson,Jing Hua Zhao,Jing Hua Zhao,Robert A. Scott,Dominique Gauguier,Dominique Gauguier,Svati H. Shah,Svati H. Shah,Albert V. Smith,Natalie van Zuydam,Amanda J. Cox,Christina Willenborg,Christina Willenborg,Christina Willenborg,Thorsten Kessler,Lingyao Zeng,Michael A. Province,Andrea Ganna,Lars Lind,Nancy L. Pedersen,Charles C. White,Anni Joensuu,Marcus Edi Kleber,Alistair S. Hall,Winfried März,Veikko Salomaa,Christopher J. O'Donnell,Erik Ingelsson,Mary F. Feitosa,Jeanette Erdmann,Donald W. Bowden,Donald W. Bowden,Colin N. A. Palmer,Colin N. A. Palmer,Vilmundur Gudnason,Ulf de Faire,Pierre Zalloua,Nicholas J. Wareham,John Thompson,Kari Kuulasmaa,George V. Dedoussis,George V. Dedoussis,Markus Perola,Abbas Dehghan,John C. Chambers,John C. Chambers,Jaspal S. Kooner,Hooman Allayee,Panos Deloukas,Ruth McPherson,Ruth McPherson,K. Stefansson,Heribert Schunkert,Sekar Kathiresan,Sekar Kathiresan,Martin Farrall,Philippe M. Frossard,Daniel J. Rader,Nilesh J. Samani,Muredach P. Reilly +90 more
TL;DR: In this paper, the interaction of genetic susceptibility with lifestyle factors may play a prominent role in coronary heart disease (CHD) etiology and the interaction between genetic susceptibility and lifestyle factors is discussed.