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Erik Ingelsson

Researcher at Stanford University

Publications -  546
Citations -  99427

Erik Ingelsson is an academic researcher from Stanford University. The author has contributed to research in topics: Genome-wide association study & Population. The author has an hindex of 124, co-authored 538 publications receiving 85407 citations. Previous affiliations of Erik Ingelsson include Karolinska Institutet & Cardiovascular Institute of the South.

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Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk

Helen R. Warren, +267 more
TL;DR: Results from genetic risk score models raise the possibility of a precision medicine approach through early lifestyle intervention to offset the impact of blood pressure–raising genetic variants on future cardiovascular disease risk.
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Hysterectomy and risk of cardiovascular disease: a population-based cohort study.

TL;DR: Hysterectomy in women aged 50 years or younger substantially increases the risk for CVD later in life and oopherectomy further adds to the risk of both coronary heart disease and stroke.
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Directional dominance on stature and cognition in diverse human populations

Peter K. Joshi, +358 more
- 23 Jul 2015 - 
TL;DR: This study provides evidence that increased stature and cognitive function have been positively selected in human evolution, whereas many important risk factors for late-onset complex diseases may not have been.
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Pro-efferocytic nanoparticles are specifically taken up by lesional macrophages and prevent atherosclerosis

TL;DR: A macrophage-specific nanotherapy based on single-walled carbon nanotubes loaded with a chemical inhibitor of the antiphagocytic CD47-SIRPα signalling axis is developed, demonstrating the potential of ‘Trojan horse’ nanoparticles to prevent atherosclerotic cardiovascular disease.

Association analyses of 249,796 individuals reveal 18 new loci associated with body mass index

Elizabeth K. Speliotes, +413 more
TL;DR: In this article, the authors examined associations between body mass index and similar to 2.8 million SNPs in up to 123,865 individuals with targeted follow up of 42 SNPs and up to 125,931 additional individuals.