E
Erik Ingelsson
Researcher at Stanford University
Publications - 546
Citations - 99427
Erik Ingelsson is an academic researcher from Stanford University. The author has contributed to research in topics: Genome-wide association study & Population. The author has an hindex of 124, co-authored 538 publications receiving 85407 citations. Previous affiliations of Erik Ingelsson include Karolinska Institutet & Cardiovascular Institute of the South.
Papers
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Journal ArticleDOI
Burden and prognostic importance of subclinical cardiovascular disease in overweight and obese individuals.
Erik Ingelsson,Lisa M. Sullivan,Caroline S. Fox,Caroline S. Fox,Joanne M. Murabito,Joanne M. Murabito,Emelia J. Benjamin,Emelia J. Benjamin,Joseph F. Polak,James B. Meigs,Michelle J. Keyes,Michelle J. Keyes,Christopher J. O'Donnell,Christopher J. O'Donnell,Thomas J. Wang,Thomas J. Wang,Ralph B. D'Agostino,Ralph B. D'Agostino,Philip A. Wolf,Philip A. Wolf,Ramachandran S. Vasan,Ramachandran S. Vasan +21 more
TL;DR: In this community-based sample, overweight and obesity were associated with high prevalence of subclinical disease, which partly contributed to the increased risk of overt CVD in these strata.
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Enabling Efficient and Confident Annotation of LC-MS Metabolomics Data through MS1 Spectrum and Time Prediction.
Corey D. Broeckling,Andrea Ganna,Mark Layer,Kevin Brown,Ben Sutton,Erik Ingelsson,Graham Peers,Jessica E. Prenni +7 more
TL;DR: This work reports chromatographic and mass spectrometric behavior of 904 authentic standards collected under conditions identical to a typical nontargeted profiling experiment and indicates that in-source phenomena are not random but depend at least in part on chemical structure.
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Methylation-based estimated biological age and cardiovascular disease.
TL;DR: The aim of this population‐based longitudinal cohort study was to investigate the association between estimated biological age and incident cardiovascular disease (CVD).
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Human Genetics of Obesity and Type 2 Diabetes Mellitus: Past, Present, and Future.
TL;DR: A review of the current knowledge and suggestions for the next steps in genetic research for Type 2 diabetes mellitus (T2D) and obesity can be found in this paper, where the authors focus on four areas relevant to precision medicine: genetic architecture, pharmacogenetics and other gene-environment interactions, mechanistic inference, and drug development.
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Non-targeted metabolomics combined with genetic analyses identifies bile acid synthesis and phospholipid metabolism as being associated with incident type 2 diabetes
Tove Fall,Tove Fall,Samira Salihovic,Samira Salihovic,Stefan Brandmaier,Christoph Nowak,Christoph Nowak,Andrea Ganna,Stefan Gustafsson,Stefan Gustafsson,Corey D. Broeckling,Jessica E. Prenni,Gabi Kastenmüller,Annette Peters,Patrik K. E. Magnusson,Rui Wang-Sattler,Vilmantas Giedraitis,Christian Berne,Christian Gieger,Nancy L. Pedersen,Erik Ingelsson,Erik Ingelsson,Erik Ingelsson,Lars Lind +23 more
TL;DR: Novel findings included associations of higher values of the bile acid deoxycholic acid and monoacylglyceride 18:2 and lower concentrations of cortisol with type 2 diabetes risk but adding metabolites to an existing risk score improved model fit only marginally.