F
Frank M. Sacks
Researcher at Harvard University
Publications - 520
Citations - 86842
Frank M. Sacks is an academic researcher from Harvard University. The author has contributed to research in topics: Cholesterol & Weight loss. The author has an hindex of 120, co-authored 490 publications receiving 80422 citations. Previous affiliations of Frank M. Sacks include Erasmus University Rotterdam & University of Texas Health Science Center at Houston.
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Journal ArticleDOI
Genetic Determinant for Amino Acid Metabolites and Changes in Body Weight and Insulin Resistance in Response to Weight-Loss Diets The Preventing Overweight Using Novel Dietary Strategies (POUNDS LOST) Trial
TL;DR: In this paper, the effects of a genetic determinant of branched-chain amino acid/aromatic amino acid ratio on changes in body weight and insulin resistance in a 2-year diet intervention trial were investigated.
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FTO genotype, dietary protein, and change in appetite: the Preventing Overweight Using Novel Dietary Strategies trial
Tao Huang,Qibin Qi,Yanping Li,Frank B. Hu,George A. Bray,Frank M. Sacks,Donald A. Williamson,Lu Qi +7 more
TL;DR: The data suggest that individuals with the FTO rs9939609 A allele might obtain more benefits in a reduction of food cravings and appetite by choosing a hypocaloric and higher-protein weight-loss diet.
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Plasma Lipoprotein Levels in Vegetarians: The Effect of Ingestion of Fats From Dairy Products
Frank M. Sacks,Dean Ornish,Bernard Rosner,Sandra McLanahan,William P. Castelli,Edward H. Kass +5 more
TL;DR: Analysis within and among vegetarian populations suggests that ingestion of fatty dairy products raises the LDL-C levels on a percentage basis about three times more than it raises the HDL-C level.
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Apolipoproteins E and CIII interact to regulate HDL metabolism and coronary heart disease risk
Allyson M. Morton,Manja Koch,Carlos O. Mendivil,Jeremy D. Furtado,Anne Tjønneland,Kim Overvad,Liyun Wang,Majken K. Jensen,Frank M. Sacks +8 more
TL;DR: Differences in metabolism of HDL subspecies pertaining to reverse cholesterol transport are reflected in differences in association with CHD, and HDL apoE was associated significantly with lower risk of CHD only in the HDL sub species lacking apoCIII.
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The platelet PlA2 and angiotensin-converting enzyme (ACE) D allele polymorphisms and the risk of recurrent events after acute myocardial infarction
Paul F. Bray,Christopher P. Cannon,Pascal J. Goldschmidt-Clermont,Lemuel A. Moyé,Marc A. Pfeffer,Frank M. Sacks,Eugene Braunwald +6 more
TL;DR: The Pl A1,A2 genotype was associated with an excess of recurrent coronary events in patients after MI who did not receive pravastatin, and the ACE D allele added to this risk.