G
Golo Henning
Researcher at Hannover Medical School
Publications - 4
Citations - 1323
Golo Henning is an academic researcher from Hannover Medical School. The author has contributed to research in topics: CC chemokine receptors & C-C chemokine receptor type 7. The author has an hindex of 4, co-authored 4 publications receiving 1248 citations.
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Journal ArticleDOI
CCR7 Governs Skin Dendritic Cell Migration under Inflammatory and Steady-State Conditions
Lars Ohl,Mariette Mohaupt,Niklas Czeloth,Gabriele Hintzen,Ziba Kiafard,Jörg Zwirner,Thomas Blankenstein,Golo Henning,Reinhold Förster +8 more
TL;DR: The data identify CCR7 as a key regulator that governs trafficking of skin DC under both inflammatory and steady-state conditions and provides evidence that these cells represent a semimature population of DC that is capable of initiating T cell proliferation under conditions known to induce tolerance.
Journal ArticleDOI
Cooperating mechanisms of CXCR5 and CCR7 in development and organization of secondary lymphoid organs.
Lars Ohl,Golo Henning,Stefan Krautwald,Martin Lipp,Svenja Hardtke,Giinter Bernhardt,Oliver Pabst,Reinhold Förster +7 more
TL;DR: It is demonstrated that CD3− CD4+ IL-7Rαhi cells express CX CR5 as well as CCR7 indicating that both receptors cooperate during an early step of secondary lymphoid organ development, demonstrating a cooperative function of CXCR5 and CCR 7 in lymphoidorgan organogenesis and organization.
Journal ArticleDOI
CC Chemokine Receptor 7–dependent and –independent Pathways for Lymphocyte Homing Modulation by FTY720
Golo Henning,Lars Ohl,Tobias Junt,Phillip Reiterer,Volker Brinkmann,Hideki Nakano,Werner Hohenberger,Martin Lipp,Reinhold Förster +8 more
TL;DR: The data suggest an alternative G-αi-dependent, CCR7-CCL19/CCL21-independent mechanism for lymphocyte homing through HEVs which is strongly augmented in the presence of FTY720.
Journal ArticleDOI
Lessons Learned From Lymphocytes: CC Chemokine Receptor-7 Involved in Lymphogenic Metastasis of Melanoma
TL;DR: The metastatic potential of primary tumors is the chief prognostic determinant of malignant disease and the underlying molecular mechanisms along with the alterations in gene expression of tumor cells required for these processes have begun to emerge only recently.