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Hiroaki Saito

Researcher at University of Tokyo

Publications -  182
Citations -  7536

Hiroaki Saito is an academic researcher from University of Tokyo. The author has contributed to research in topics: Phytoplankton & Bloom. The author has an hindex of 42, co-authored 171 publications receiving 6940 citations. Previous affiliations of Hiroaki Saito include National Agriculture and Food Research Organization & Japanese Ministry of Agriculture, Forestry and Fisheries.

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Synthesis of iron fertilization experiments: From the iron age in the age of enlightenment

TL;DR: In this paper, a comparison of eight iron experiments shows that maximum Chl a, the maximum DIC removal, and the overall DIC/Fe efficiency all scale inversely with depth of the wind mixed layer (WML) defining the light environment.
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The decline and fate of an iron-induced subarctic phytoplankton bloom

TL;DR: The depletion of silicic acid and the inefficient transfer of iron-increased POC below the permanent thermocline have major implications both for the biogeochemical interpretation of times of greater iron supply in the geological past, and also for proposed geo-engineering schemes to increase oceanic carbon sequestration.
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Iron supply to the western subarctic Pacific: Importance of iron export from the Sea of Okhotsk

TL;DR: This article found extremely high concentrations of dissolved and particulate iron in the Okhotsk Sea Intermediate Water and the North Pacific Intermediate Water (NPIW) and showed that water ventilation processes in this region probably control the transport of iron through the intermediate water layer from the continental shelf of the Sea of Ohotsk to wide areas of the western subarctic Pacific (WSP).
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Docosahexaenoic acid suppresses nitric oxide production and inducible nitric oxide synthase expression in interferon-γ plus lipopolysaccharide-stimulated murine macrophages by inhibiting the oxidative stress

TL;DR: It is demonstrated that DHA inhibits NO production in macrophages and this inhibition is, in part, mediated by upregulation of GSH, which is thought to mediate the activity of DHA.