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Jean-Pierre Gorvel

Researcher at Aix-Marseille University

Publications -  239
Citations -  16211

Jean-Pierre Gorvel is an academic researcher from Aix-Marseille University. The author has contributed to research in topics: Brucella & Endosome. The author has an hindex of 67, co-authored 231 publications receiving 15005 citations. Previous affiliations of Jean-Pierre Gorvel include Centre national de la recherche scientifique & Aberystwyth University.

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Bacterial infection and non-Hodgkin’s lymphoma

TL;DR: Evidence in favour of a link between Helicobacter pylori, Chlamydia psittaci, Coxiella burnetii, Borrelia burgdorferi and Campylobacter jejuni infection and non-Hodgkin’s lymphoma is reviewed.
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An immunologist's look at the Rho and Rab GTP-binding proteins

TL;DR: The subject of this review is to outline briefly the areas of lymphocyte function which may implicate small G proteins, with special emphasis on the established or possible roles of proteins of the Rho and Rab subfamilies in cytoskeleton organization and antigen presentation.
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Recognition of sodium- and potassium-dependent adenosine triphosphatase in organs of the mouse by means of a monoclonal antibody

TL;DR: Evidence is presented that this rat anti-mouse monoclonal antibody (m Ab), anti-BSP-3, recognizes mouse (Na++K+)-ATPase (ATP phosphohydrolase, E.C.3.6.3).
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Characterization of the neutral aminopeptidase activity associated to the mouse thymocyte-activating molecule.

TL;DR: It is shown that the THAM-specific mAb H194-112 exhibits strong reactivity with several nonlymphoid tissues including polarized enterocytes from small intestine, kidney cortical tubuli, and liver bile ductuli, as well as kidney glomeruli and lung alveoloar pneumocytes.
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Brucella abortus induces Irgm3 and Irga6 expression via type-I IFN by a MyD88-dependent pathway, without the requirement of TLR2, TLR4, TLR5 and TLR9.

TL;DR: It is shown in murine macrophages that Brucella abortus triggers expression of the interferon-inducible resistance proteins (IRGs, p47 GTPases) via type-I IFN secretion at late time points, when Bru Cella has reached its replication niche.