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Katja Wassmann

Researcher at University of Paris

Publications -  44
Citations -  2997

Katja Wassmann is an academic researcher from University of Paris. The author has contributed to research in topics: Chromosome segregation & Cohesin. The author has an hindex of 24, co-authored 37 publications receiving 2698 citations. Previous affiliations of Katja Wassmann include French Institute of Health and Medical Research & Hannover Medical School.

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The Mad2 spindle checkpoint protein has two distinct natively folded states.

TL;DR: It is shown that without cofactor binding or covalent modification Mad2 adopts two distinct folded conformations at equilibrium (termed N1-Mad2 and N2-Mad1), suggesting that the unusual two-state behavior of Mad2 is critical for spindle checkpoint signaling.
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Resolution of chiasmata in oocytes requires separase-mediated proteolysis.

TL;DR: By expressing Cre recombinase from a zona pellucida promoter, a floxed allele of separase is deleted and prevents removal of Rec8 from chromosome arms and resolution of chiasmata, which hinders extrusion of the first polar body (PBE) and causes female sterility.
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Metaphase I Arrest upon Activation of the Mad2-Dependent Spindle Checkpoint in Mouse Oocytes

TL;DR: This study shows for the first time that a functional Mad2-dependent spindle checkpoint exists during the first meiotic division in mammalian oocytes and investigates the mechanisms ensuring correct separation of chromosomes in meiosis I.
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Meiotic homologue alignment and its quality surveillance are controlled by mouse HORMAD1

TL;DR: This work shows that mouse HORMAD1 ensures that sufficient numbers of processed DSBs are available for successful homology search, and proposes that the synaptonemal complex and HORMad1 are key components of a negative feedback loop that coordinates meiotic progression with homologue alignment.
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Mitotic checkpoints: from yeast to cancer.

TL;DR: Molecular mechanisms controlling this checkpoint have been identified and loss of this checkpoint has been shown to result in chromosome missegregation in higher eukaryotes and may contribute to the genomic instability observed in human cancers.