K
Katsuhisa Ikeda
Researcher at Juntendo University
Publications - 456
Citations - 7596
Katsuhisa Ikeda is an academic researcher from Juntendo University. The author has contributed to research in topics: Cochlea & Sinusitis. The author has an hindex of 43, co-authored 445 publications receiving 6922 citations. Previous affiliations of Katsuhisa Ikeda include Tohoku University & University of Minnesota.
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Journal ArticleDOI
Novel scoring system and algorithm for classifying chronic rhinosinusitis: the JESREC Study
Takahiro Tokunaga,Masafumi Sakashita,Takenori Haruna,Daiya Asaka,Sachio Takeno,H. Ikeda,Tsuguhisa Nakayama,Nobuhiko Seki,S. Ito,Junko Murata,Yasunori Sakuma,Naohiro Yoshida,Tetsuya Terada,Ichiro Morikura,Hiroshi Sakaida,Kenji Kondo,K. Teraguchi,Mitsuhiro Okano,Nobuyoshi Otori,Mamoru Yoshikawa,Katsuhiro Hirakawa,Shinichi Haruna,Tetsuo Himi,Katsuhisa Ikeda,Junichi Ishitoya,Yukiko Iino,Ryo Kawata,Hideyuki Kawauchi,Masayoshi Kobayashi,Tatsuya Yamasoba,Takaki Miwa,Mitsuyoshi Urashima,Mayumi Tamari,Emiko Noguchi,Takahiro Ninomiya,Takahiro Ninomiya,Yoshimasa Imoto,Taiyo Morikawa,Kaori Tomita,Tetsuji Takabayashi,Shigeharu Fujieda +40 more
TL;DR: This study wanted to set objective clinical criteria for the diagnosis of refractory CRS, and found that CRSwNP displays more intense eosinophilic infiltration and the presence of Th2 cytokines.
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Novel mutations in the connexin 26 gene (GJB2) responsible for childhood deafness in the Japanese population.
Takayuki Kudo,Katsuhisa Ikeda,Shigeo Kure,Yoichi Matsubara,Takeshi Oshima,Kenichi Watanabe,Tetsuaki Kawase,Kuniaki Narisawa,Tomonori Takasaka +8 more
TL;DR: Findings indicate that GJB2 mutations are also responsible for prelingual deafness in Japan.
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Altered Cochlear Fibrocytes in a Mouse Model of DFN3 Nonsyndromic Deafness
Osamu Minowa,Katsuhisa Ikeda,Yoshinobu Sugitani,Takeshi Oshima,Shigeyasu Nakai,Yukio Katori,Masaaki Suzuki,Masayuki Furukawa,Tetsuaki Kawase,Y. Zheng,Masaki Ogura,Yukinori Asada,Kenichi Watanabe,Hitomi Yamanaka,S. Gotoh,S. Gotoh,M. Nishi-Takeshima,Tetsuo Sugimoto,Toshiaki Kikuchi,Tomonori Takasaka,Tetsuo Noda,Tetsuo Noda +21 more
TL;DR: The findings suggest that cochlear spiral ligament fibrocytes, which are mesenchymal in origin and for which a role in potassium ion homeostasis has been postulated, may play a critical role in auditory function.
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Transgenic expression of a dominant-negative connexin26 causes degeneration of the organ of Corti and non-syndromic deafness
Takayuki Kudo,Shigeo Kure,Katsuhisa Ikeda,An Ping Xia,Yukio Katori,Masaaki Suzuki,Kanako Kojima,Akiko Ichinohe,Yoichi Suzuki,Yoko Aoki,Toshimitsu Kobayashi,Yoichi Matsubara +11 more
TL;DR: The results suggest that the GJB2 mutation disturbs homeostasis of cortilymph, an extracellular space surrounding the sensory hair cells, due to impaired K(+) transport by supporting cells, resulting in degradation of the organ of Corti, rather than affecting endolymphHomeostasis in mice and probably in humans.
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Cisplatin-induced apoptotic cell death in Mongolian gerbil cochlea.
Shaheen Ara Alam,Katsuhisa Ikeda,Takeshi Oshima,Masaaki Suzuki,Tetsuaki Kawase,Toshihiko Kikuchi,Tomonori Takasaka +6 more
TL;DR: A critical role is suggested for bcl-2 family proteins in the regulation of apoptotic cell death induced by cisplatin and a significant deterioration in the responses of both distortion product otoacoustic emissions and the endocochlear potential are suggested.