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Mark W. Russell

Researcher at University of Michigan

Publications -  130
Citations -  5870

Mark W. Russell is an academic researcher from University of Michigan. The author has contributed to research in topics: Hypertrophic cardiomyopathy & Obscurin. The author has an hindex of 37, co-authored 118 publications receiving 4585 citations. Previous affiliations of Mark W. Russell include Boston Children's Hospital & Case Western Reserve University.

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De novo mutations in congenital heart disease with neurodevelopmental and other congenital anomalies.

TL;DR: Clinical genotyping of patients with CHD may help to identify those at greatest risk of neurodevelopmental disabilities, allowing surveillance and early intervention and provide opportunities for improved prognostic assessment and early therapeutic intervention in CHD patients.
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Contribution of rare inherited and de novo variants in 2,871 congenital heart disease probands

TL;DR: Exome sequencing of a single cohort of 2,871 CHD probands, including 2,645 parent–offspring trios, implicated rare inherited mutations in 1.8%, and DNMs in ∼440 genes were inferred to contribute to CHD.
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Genetic Basis for Congenital Heart Disease: Revisited: A Scientific Statement From the American Heart Association.

TL;DR: This review provides an updated summary of the state of the authors' knowledge of the genetic contributions to the pathogenesis of congenital heart disease and recent advances in the understanding of copy number variants, syndromes, RASopathies, and heterotaxy/ciliopathies.
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Cardiomyocyte-Specific Knockout and Agonist of Peroxisome Proliferator–Activated Receptor-γ Both Induce Cardiac Hypertrophy in Mice

TL;DR: It is shown that cardiomyocyte PPAR-γ suppresses cardiac growth and embryonic gene expression and inhibits nuclear factor &kgr;B activity in vivo and rosiglitazone causes cardiac hypertrophy at least partially independent of PPar-γ inCardiomyocytes and through different mechanisms from CM-PGKO.