M
Michael C. Neale
Researcher at Virginia Commonwealth University
Publications - 647
Citations - 72612
Michael C. Neale is an academic researcher from Virginia Commonwealth University. The author has contributed to research in topics: Twin study & Population. The author has an hindex of 121, co-authored 620 publications receiving 66343 citations. Previous affiliations of Michael C. Neale include VU University Amsterdam & University of East London.
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Multivariate genetic analysis of twin-family data on fears: Mx models.
TL;DR: The implementation of multivariate models of familial resemblance with the Mx package allows for the effects of assortative mating, additive and dominant genes, common and specific environment, and both genetic and cultural transmission between generations.
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The clinical characteristics of major depression as indices of the familial risk to illness.
TL;DR: The results suggest that the clinical features of MD can be meaningfully related to the familial vulnerability to illness, particularly with respect to recurrence, impairment and patterns of co-morbidity.
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Bias in correlations from selected samples of relatives: the effects of soft selection.
TL;DR: An alternative model of soft selection is presented which has strikingly different consequences for the resemblance between relatives and the softer the threshold, the more the correlation resembles that in the underlying population.
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Longitudinal genetic analysis of problem behaviors in biologically related and unrelated adoptees
TL;DR: The genetic and environmental influences on problem behaviors at two assessment points, three years apart, and their stability were studied in a sample of international adoptees, initially aged 10 to 15 years.
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The structure of genetic and environmental risk factors for three measures of disordered eating.
Tracey D. Wade,Nicholas G. Martin,Michael C. Neale,Marika Tiggemann,Susan A. Treloar,K. K. Bucholz,P. A. F. Madden,A. C. Heath +7 more
TL;DR: Lability to the development of the behaviours and attitudes characteristic of eating disorders is best explained by both environmental and genetic factors, with covariation between the three measures best explainedBy a single latent phenotype of disordered eating which has a heritability of 60%.