Michael C. Neale

TL;DR: The genes and individual-specific experiences that predispose to premenstrual symptoms appear to be largely distinct from those which predispose either to menstrual or to neurotic symptoms.

TL;DR: In this article, the non-synonymous variant of the mu1 opioid receptor gene (A118G, Asn40Asp) has been extensively studied, yet its role in addiction has remained unclear, with conflicting association findings.

TL;DR: Phenotypic and genetic associations of cortisol levels with the thickness of prefrontal and anterior cingulate cortex regions, and with hippocampal volume in a sample of 388 middle-aged male twins who were 51-59 years old remained significant after adjusting for general cognitive ability, cardiovascular risk factors, and depression.

TL;DR: It is suggested that genetic factors play a moderate etiologic role in the 1YP-MD, the temporal stability of the liability to major depression in adult women is largely or entirely genetic in origin, and environmental Factors play a significant role inThe etiology of major depression, but their effects are generally transitory and do not result in enduring changes in the liabilities to illness.

TL;DR: Dynamic modeling of changes in heritability over time demonstrated that the heritability of cortical thickness increases gradually throughout late childhood and adolescence, with sequential emergence of three large regions of high heritability in the temporal poles, the inferior parietal lobes, and the superior and dorsolateral frontal cortices.