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Michael C. Neale

Researcher at Virginia Commonwealth University

Publications -  647
Citations -  72612

Michael C. Neale is an academic researcher from Virginia Commonwealth University. The author has contributed to research in topics: Twin study & Population. The author has an hindex of 121, co-authored 620 publications receiving 66343 citations. Previous affiliations of Michael C. Neale include VU University Amsterdam & University of East London.

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Testosterone modifies the effect of APOE genotype on hippocampal volume in middle-aged men

TL;DR: These findings demonstrate an interaction effect between testosterone and the APOE ε4 allele on hippocampal volume in middle-aged men, and they may suggest 2 low testosterone subgroups, allude to potential gene–gene interactions between APOE and either androgen receptor polymorphisms or genes associated with testosterone production.
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Perceived support and adjustment to stress in a general population sample of female twins.

TL;DR: The stress-buffering effect of perceived support is explored in a large panel survey of adult female twins and no evidence was found for any of the hypotheses investigated to explain the interaction between perceived support and acute stress.
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Frequency of church attendance in Australia and the United States: models of family resemblance.

TL;DR: Both cohorts of twins and their families from the USA and Australia indicated significant additive genetic and shared environment effects on church attendance, with minor contributions from twin environment, assortative mating and parent-offspring environmental transmission.
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A longitudinal twin study of borderline and antisocial personality disorder traits in early to middle adulthood.

TL;DR: ASD and BPD traits were moderately stable from early to middle adulthood, mostly due to genetic risk factors which did not change over the 10-year assessment period, and environmental risk factors were mostly transient, and appear to be the main source of phenotypic change.
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Assessing the heritability of anorexia nervosa symptoms using a marginal maximal likelihood approach.

TL;DR: AN symptoms are differentially heritable, and specific criteria such as those related to body weight and weight loss history represent more biologically driven potential endophenotypes or liability indices.