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Michael R. McLaren

Researcher at North Carolina State University

Publications -  20
Citations -  1244

Michael R. McLaren is an academic researcher from North Carolina State University. The author has contributed to research in topics: Microbiome & Metagenomics. The author has an hindex of 10, co-authored 19 publications receiving 855 citations. Previous affiliations of Michael R. McLaren include University of Pennsylvania & Stanford University.

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The Atacama Cosmology Telescope: Cosmological Parameters from Three Seasons of Data

Jonathan Sievers, +115 more
TL;DR: In this article, a model of primary cosmological and secondary foreground parameters is fit to the map power spectra and lensing deflection power spectrum, including contributions from both the thermal Sunyaev-Zeldovich (tSZ) effect and the kinematic SZ effect, Poisson and correlated anisotropy from unresolved infrared sources, radio sources and the correlation between the tSZ effect and infrared sources.
Journal ArticleDOI

Consistent and correctable bias in metagenomic sequencing experiments.

TL;DR: This work proposes a mathematical model for how bias distorts community measurements based on the properties of real experiments and validate this model with 16S rRNA gene and shotgun metagenomics data from defined bacterial communities.
Posted ContentDOI

Consistent and correctable bias in metagenomic sequencing measurements

TL;DR: This work proposes a mathematical model for how bias distorts community measurements based on the properties of real experiments and validate this model with 16S rRNA gene and shotgun metagenomics data from defined bacterial communities.
Journal ArticleDOI

The Atacama Cosmology Telescope: Detection of Sunyaev-Zel'Dovich Decrement in Groups and Clusters Associated with Luminous Red Galaxies

TL;DR: In this paper, a detection of the Sunyaev-Zel'dovich (SZ) decrement associated with the Luminous Red Galaxy (LRG) sample of the Sloan Digital Sky Survey is presented.
Journal ArticleDOI

Clostridioides difficile exploits toxin-mediated inflammation to alter the host nutritional landscape and exclude competitors from the gut microbiota

TL;DR: In this paper, the authors investigate how toxin-induced inflammation alters C. difficile metabolism, tissue gene expression and the gut microbiota, and determine how inflammation by the host may be beneficial to C. Difficile.