P
Perla Kaliman
Researcher at Open University of Catalonia
Publications - 48
Citations - 2679
Perla Kaliman is an academic researcher from Open University of Catalonia. The author has contributed to research in topics: Myogenesis & Skeletal muscle. The author has an hindex of 26, co-authored 47 publications receiving 2338 citations. Previous affiliations of Perla Kaliman include University of Wisconsin-Madison & University of Barcelona.
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Journal ArticleDOI
Epicardial retinoid X receptor is required for myocardial growth and coronary artery formation
Esther Merki,Monica Zamora,Monica Zamora,Angel Raya,Yasuhiko Kawakami,Jianming Wang,Xiaoxue Zhang,John B.E. Burch,Steven W. Kubalak,Perla Kaliman,Juan Carlos Izpisua Belmonte,Kenneth R. Chien,Kenneth R. Chien,Pilar Ruiz-Lozano,Pilar Ruiz-Lozano +14 more
TL;DR: It is shown that RXR α signaling in the epicardium is required for proper cardiac morphogenesis and an additional phenotype of defective coronary arteriogenesis associated with RXRα deficiency is detected and a retinoid-dependent Wnt signaling pathway that cooperates in epicardial epithelial-to-mesenchymal transformation is identified.
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APJ acts as a dual receptor in cardiac hypertrophy
Maria Cecilia Scimia,Cecilia Hurtado,Saugata Ray,Scott A. Metzler,Ke Wei,Jianming Wang,Christopher E. Woods,Nicole H. Purcell,Daniele Catalucci,Takeshi Akasaka,Orlando F. Bueno,George P. Vlasuk,Perla Kaliman,Rolf Bodmer,Layton H. Smith,Euan A. Ashley,Mark Mercola,Joan Heller Brown,Pilar Ruiz-Lozano,Pilar Ruiz-Lozano +19 more
TL;DR: It is reported that genetic loss of APJ, a G-protein-coupled receptor, confers resistance to chronic pressure overload by markedly reducing myocardial hypertrophy and heart failure, and indicates that APJ is a bifunctional receptor for both mechanical stretch and the endogenous peptide apelin.
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Rapid changes in histone deacetylases and inflammatory gene expression in expert meditators
Perla Kaliman,María Jesús Álvarez-López,Marta Cosín-Tomás,Melissa A. Rosenkranz,Antoine Lutz,Antoine Lutz,Richard J. Davidson +6 more
TL;DR: The regulation of HDACs and inflammatory pathways may represent some of the mechanisms underlying the therapeutic potential of mindfulness-based interventions and set the foundation for future studies to further assess meditation strategies for the treatment of chronic inflammatory conditions.
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Phosphatidylinositol 3-kinase inhibitors block differentiation of skeletal muscle cells.
TL;DR: Data indicate that whereas phosphatidylinositol 3-kinase is not indispensable for cell proliferation or in the initial events of myoblast differentiation, it appears to be essential for terminal differentiation of muscle cells.
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Histone demethylase LSD1 regulates adipogenesis
TL;DR: The findings indicate that the histone methylation status of adipogenic genes as well as the expression and function of the proteins involved in its maintenance play a crucial role in adipogenesis.