P
Petronela Ancuta
Researcher at Université de Montréal
Publications - 90
Citations - 9116
Petronela Ancuta is an academic researcher from Université de Montréal. The author has contributed to research in topics: Proinflammatory cytokine & Medicine. The author has an hindex of 32, co-authored 82 publications receiving 8005 citations. Previous affiliations of Petronela Ancuta include French Institute of Health and Medical Research & Harvard University.
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Journal ArticleDOI
Nomenclature of monocytes and dendritic cells in blood.
Loems Ziegler-Heitbrock,Petronela Ancuta,Suzanne M. Crowe,Marc Dalod,Marc Dalod,Veronika Grau,Derek N.J. Hart,Pieter J. M. Leenen,Yong-Jun Liu,G. Gordon MacPherson,Gwendalyn J. Randolph,Juergen E. Scherberich,Juergen Schmitz,Ken Shortman,Silvano Sozzani,Herbert Strobl,Marek Zembala,Jonathan M. Austyn,Manfred B. Lutz +18 more
TL;DR: The present document proposes a nomenclature for monocytes and defines 3 types of monocytes (classical, intermediate, and nonclassical monocytes) and3 types of dendritic cells (plasmacytoid and 2 types of myeloid dendrites) in human and in mouse blood.
Journal ArticleDOI
HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation
Nicolas Chomont,Mohamed El-Far,Petronela Ancuta,Lydie Trautmann,Francesco A. Procopio,Bader Yassine-Diab,Geneviève Boucher,Mohamed Rachid Boulassel,Georges Ghattas,Jason M. Brenchley,Timothy W. Schacker,Brenna J. Hill,Daniel C. Douek,Jean-Pierre Routy,Elias K. Haddad,Elias K. Haddad,Rafick Pierre Sekaly +16 more
TL;DR: The results suggest that viral eradication might be achieved through the combined use of strategic interventions targeting viral replication and drugs that interfere with the self renewal and persistence of proliferating memory T cells.
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Fractalkine Preferentially Mediates Arrest and Migration of CD16+ Monocytes
Petronela Ancuta,Ravi M. Rao,Ashlee V. Moses,Andrew Mehle,Sunil K. Shaw,F. William Luscinskas,Dana Gabuzda +6 more
TL;DR: It is demonstrated that FKN preferentially mediates arrest and migration of CD16+ monocytes and suggested that recruitment of this proinflammatory monocyte subset to vessel walls via the CX3CR1-FKN pathway may contribute to vascular and tissue injury during pathological conditions.
Journal ArticleDOI
Vif Overcomes the Innate Antiviral Activity of APOBEC3G by Promoting Its Degradation in the Ubiquitin-Proteasome Pathway
TL;DR: It is reported here that Vif counteracts the antiviral activity of APOBEC3G by targeting it for destruction by the ubiquitin-proteasome pathway and implicate the proteasome as a site of dynamic interplay between microbial and cellular defenses.
Journal ArticleDOI
Microbial translocation is associated with increased monocyte activation and dementia in AIDS patients.
Petronela Ancuta,Anupa Kamat,Kevin J. Kunstman,Eun Young Kim,Patrick Autissier,Alysse G. Wurcel,Tauheed Zaman,David Stone,Megan Mefford,Susan Morgello,Elyse J. Singer,Steven M. Wolinsky,Dana Gabuzda +12 more
TL;DR: A role for elevated LPS levels in driving monocyte activation in AIDS is suggested, thereby contributing to the pathogenesis of HAD, and evidence that cofactors linked to substance abuse and HCV co-infection influence these processes is provided.