R
Roland E. Schmieder
Researcher at University of Erlangen-Nuremberg
Publications - 780
Citations - 85811
Roland E. Schmieder is an academic researcher from University of Erlangen-Nuremberg. The author has contributed to research in topics: Blood pressure & Essential hypertension. The author has an hindex of 97, co-authored 717 publications receiving 78138 citations. Previous affiliations of Roland E. Schmieder include Complutense University of Madrid & University of Regensburg.
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Is the decrease in arterial pressure the sole factor for reduction of left ventricular hypertrophy
TL;DR: An analysis of the underlying pathogenic mechanisms suggests the presence of multiple interacting pathogenic factors in the development of left ventricular hypertrophy and disparate rates of reduction with various antihypertensive drugs point to the existence of blood pressure-independent factors influencing reduction of LVH.
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Low-dose eplerenone decreases left ventricular mass in treatment-resistant hypertension.
Andreas Schneider,Johannes Schwab,Marina V. Karg,Tatjana Kalizki,Annemarie Reinold,Markus P. Schneider,Roland E. Schmieder,Bernhard Schmidt +7 more
TL;DR: The data suggest that in patients with TRH, mineralocorticoid receptor antagonists should be used preferentially in order to achieve an effective reduction of LVM along with the improvement of BP control.
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Effect of Heart Rate on the Outcome of Renal Denervation in Patients With Uncontrolled Hypertension
Michael Böhm,Konstantinos Tsioufis,David E. Kandzari,Kazuomi Kario,Michael A. Weber,Roland E. Schmieder,Raymond R. Townsend,Saarraaken Kulenthiran,Christian Ukena,Stuart J. Pocock,Sebastian Ewen,Joachim Weil,Martin Fahy,Felix Mahfoud +13 more
TL;DR: In this article, the impact of baseline heart rate on BP reduction without antihypertensive medications in the SPYRAL HTN-OFF MED (Global Clinical Study of Renal Denervation With the Symplicity Spyral Multi-electrode Renal denervation System in Patients With Uncontrolled Hypertension in the Absence of Antihypertensive Medications) Pivotal trial was evaluated.
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MDR1 genotype-dependent regulation of the aldosterone system in humans.
TL;DR: It is demonstrated that the ABCB1 3435 genotype affects angiotensin II- Stimulated serum aldosterone levels and salt-stimulated urinary sodium excretion, indicating a new link between MDR1 genotype and the ald testosterone system in humans.
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Angiotensin converting enzyme inhibitors. Disparities in the mechanism of their antihypertensive effect.
Guillermo E. Garavaglia,Franz H. Messerli,Boris D. Nunez,Roland E. Schmieder,Edward D. Frohlich +4 more
TL;DR: Enalapril induced a twofold greater reduction than captopril (14%) or lisinopril(12%) and renal hemodynamic effects may be drug specific and not uniform for all ACE inhibitors.