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Roland E. Schmieder

Researcher at University of Erlangen-Nuremberg

Publications -  780
Citations -  85811

Roland E. Schmieder is an academic researcher from University of Erlangen-Nuremberg. The author has contributed to research in topics: Blood pressure & Essential hypertension. The author has an hindex of 97, co-authored 717 publications receiving 78138 citations. Previous affiliations of Roland E. Schmieder include Complutense University of Madrid & University of Regensburg.

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Effects of telmisartan and ramipril on adiponectin and blood pressure in patients with type 2 diabetes.

TL;DR: Adiponectin is correlated with blood pressure in patients with type 2 diabetes and whether increased adip onectin contributes to the blood pressure-lowering effect of telmisartan needs further study.
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Comparison of diastolic left ventricular filling and cardiac dysrhythmias in hypertensive patients with and without isolated septal hypertrophy

TL;DR: Age, sex, race, obesity indexes, preload, ejection fraction, and LV contractility were similar in all 4 groups, and arterial pressure and afterload were statistically similar in the 3 hypertensive groups.
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Plasma soluble adhesion molecules and endothelium-dependent vasodilation in early human atherosclerosis.

TL;DR: In hypercholesterolaemic patients without clinical atherosclerosis, levels of soluble adhesion molecules were not elevated in comparison with healthy controls, and markers of endothelial inflammation were not related to impaired endothelium-dependent vasodilation.
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Is a shared decision-making approach effective in improving hypertension management?

TL;DR: In this paper, the authors assessed whether patient empowerment in the management of hypertension improved more with the practice of shared decision making (SDM) than by education programs, and they found that SDM did not improve management when compared to an education program, which is much easier to implement in general practice.
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CYP3A5 genotype is associated with elevated blood pressure.

TL;DR: The data, as generated with a homogenous population of young Caucasians, indicate that the CYP3A5 genotype affects blood pressure in humans possibly by genotype-dependent differences in renal, CYP 3A5-mediated metabolism of cortisol and/or aldosterone.