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Sug Hyung Lee

Researcher at Catholic University of Korea

Publications -  463
Citations -  23933

Sug Hyung Lee is an academic researcher from Catholic University of Korea. The author has contributed to research in topics: Frameshift mutation & Germline mutation. The author has an hindex of 64, co-authored 454 publications receiving 21552 citations. Previous affiliations of Sug Hyung Lee include Chung-Ang University & The Catholic University of America.

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Tight Junction-Related CLDN5 and CLDN6 Genes, and Gap Junction-Related GJB6 and GJB7 Genes Are Somatically Mutated in Gastric and Colorectal Cancers.

TL;DR: The results show that CLDN5, CLDN6, GJB6 and GJB7 genes harbor not only frameshift mutations but also mutational ITH, which together may be features of GC and CRC with MSI-H.
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BH3 domain mutation of proapoptotic genes Bad, Bmf and Bcl-G is rare in transitional cell carcinomas of the urinary bladder.

TL;DR: The data presented here indicate that BH3 domain mutation of these genes is rare in TCCs and may not contribute to the pathogenesis of T CCs.
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Frameshift mutations of ATBF1, WNT9A, CYLD and PARK2 in gastric and colorectal carcinomas with high microsatellite instability

TL;DR: This study described an additional case of PAMT of the stomach, which shares the clinical, histological, immunophenotypical and ultrastructural features of the previously described cases.
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Identification of large-scale molecular changes of Autotaxin(ENPP2) knock-down by small interfering RNA in breast cancer cells.

TL;DR: The data suggest that the molecular signature identified by the ENPP2-silencing methods may represent potential candidates that can explain the complicated characteristics of ATX and may serve as biomarkers, for the development of molecular-targeting therapy, in human breast cancer.
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Protein Losing Enteropathy Associated with Henoch-Schonlein Purpura in a Patient with Rheumatoid Arthritis

TL;DR: A case of a 46 year old patient with rheumatoid arthritis presenting with Henoch-Schönlein purpura associated with protein losing enteropathy is reported.