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Timothy L. Ratliff

Researcher at Purdue University

Publications -  312
Citations -  20541

Timothy L. Ratliff is an academic researcher from Purdue University. The author has contributed to research in topics: Prostate cancer & Cancer. The author has an hindex of 69, co-authored 306 publications receiving 19092 citations. Previous affiliations of Timothy L. Ratliff include University of Iowa Hospitals and Clinics & Beth Israel Deaconess Medical Center.

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Journal ArticleDOI

The secreted antigens of Mycobacterium tuberculosis and their relationship to those recognized by the available antibodies

C. Abou-Zeid, +5 more
- 01 Feb 1988 - 
TL;DR: The present findings suggest that there are major secreted antigens to which antibodies do not yet appear to have been produced experimentally, and one of the secreted proteins has the interesting property of binding to fibronectin.
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Intravesical Bacillus Calmette-Guerin Therapy for Superficial Bladder Cancer: Effect of Bacillus Calmette-Guerin Viability on Treatment Results

David R. Kelley, +7 more
- 01 Jul 1985 - 
TL;DR: Favorable results occurred more frequently among patients who exhibited a granulomatous inflammatory response in the bladder and delayed hypersensitivity skin test response to purified protein derivative and the level of viability of each lot of bacillus Calmette-Guerin vaccine should be verified before clinical use.
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Detection of interleukin 2 in the urine of patients with superficial bladder tumors after treatment with intravesical BCG.

Eric O. Haaff, +2 more
- 01 Oct 1986 - 
TL;DR: Results show that intravesical BCG therapy induces the production of lymphokine produced in response to BCG, including IL-2, and the presence of BCG-induced lymphokines may be associated with anti-tumor activity.
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In vivo suppressive function of myeloid‐derived suppressor cells is limited to the inflammatory site

Jessica M. Haverkamp, +5 more
- 01 Mar 2011 - 
TL;DR: MDSC are important regulators of immune responses in the prostate during acute inflammation and the chronic inflammatory setting of tumor growth, and that regulation of T‐cell function by MDSC during a localized inflammatory response is restricted in vivo to the site of an ongoing immune response.

Abrogating cholesterol esterification suppresses growth and metastasis of pancreatic cancer

Junjie Li, +10 more
TL;DR: Abrogation of cholesterol esterification, either by an ACAT-1 inhibitor or by shRNA knockdown, significantly suppressed tumor growth and metastasis in an orthotopic mouse model of pancreatic cancer.