Y
Yuval Bdolah
Researcher at Hebrew University of Jerusalem
Publications - 25
Citations - 3045
Yuval Bdolah is an academic researcher from Hebrew University of Jerusalem. The author has contributed to research in topics: Preeclampsia & Embryo transfer. The author has an hindex of 17, co-authored 25 publications receiving 2763 citations. Previous affiliations of Yuval Bdolah include Beth Israel Deaconess Medical Center & Harvard University.
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Journal ArticleDOI
Soluble endoglin contributes to the pathogenesis of preeclampsia.
Shivalingappa Venkatesha,Mourad Toporsian,Chun Lam,Jun-ichi Hanai,Tadanori Mammoto,Yeon Mee Kim,Yeon Mee Kim,Yuval Bdolah,Kee-Hak Lim,Hai Tao Yuan,Towia A. Libermann,Isaac E. Stillman,Drucilla J. Roberts,Patricia A. D'Amore,Franklin H. Epstein,Frank W. Sellke,Roberto Romero,Roberto Romero,Vikas P. Sukhatme,Michelle Letarte,S. Ananth Karumanchi +20 more
TL;DR: A novel placenta-derived soluble TGF-β coreceptor, endoglin (sEng), which is elevated in the sera of preeclamptic individuals, correlates with disease severity and falls after delivery, suggest that sEng may act in concert with sFlt1 to induce severe preeclampsia.
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Endometrial NK Cells Are Special Immature Cells That Await Pregnancy
Irit Manaster,Sa'ar Mizrahi,Debra Goldman-Wohl,Hen Y. Sela,Noam Stern-Ginossar,Dikla Lankry,Raizy Gruda,Arye Hurwitz,Yuval Bdolah,Ronit Haimov-Kochman,Simcha Yagel,Ofer Mandelboim +11 more
TL;DR: It is suggested that eNK cells are inactive cells that are present in the endometrium before conception, waiting for pregnancy.
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Hypoxia and sFlt-1 in Preeclampsia: The “Chicken-and-Egg” Question
TL;DR: New evidence is provided that effects of hypoxia are cell type specific and that in placental cytotrophoblasts, hypoxIA induces an excess production of sFlt-1, which leads to VEGF deficiency and, consequently, an antiangiogenic state.
Journal ArticleDOI
Twin pregnancy and the risk of preeclampsia: bigger placenta or relative ischemia?
Yuval Bdolah,Yuval Bdolah,Chun Lam,Augustine Rajakumar,Venkatesha Shivalingappa,Walter P. Mutter,Benjamin P. Sachs,Kee-Hak Lim,Tali Bdolah-Abram,Franklin H. Epstein,S. Ananth Karumanchi +10 more
TL;DR: The findings suggest that the increased risk of preeclampsia in twin pregnancies may be due to increased placental mass that leads to increased circulating levels of sFlt1.
Journal ArticleDOI
Angiogenic imbalance in the pathophysiology of preeclampsia: newer insights.
TL;DR: Accumulating recent evidence is shown that the maternal clinical syndrome of preeclampsia is an antiangiogenic state resulting from an excess of anti-endothelial factors liberated by the diseased placenta.